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Researchers created a mouse model for Cushing's syndrome to study glucocorticoid excess and potential treatments.

Cupric chloride treatment corrected abnormal Purkinje cell development in brindled mice.

Ets2 gene is crucial for placental development in mice.

Brindled mice show abnormal catecholamine neuron development due to copper deficiency.

A position effect on the TRPS1 gene causes excessive hair growth in humans and mice.

Removing Sprouty genes in mice causes various hormone-related issues but does not increase cancer risk by one year of age.

Mutant MK6a transgenes in mice cause blistering, hair loss, and potential human alopecia.

HtrA2 activity is crucial for normal hair growth by regulating fat cell development.

Transferrin receptor 1 is essential for normal function in the intestines, pancreas, and skin.

A new mutation in the Hr gene causes hair loss in mice, similar to a human hair disorder.

Alopecia in VDR knockout mice is due to a defect in hair cycle initiation, not keratinocyte issues.

Introducing the rat OTC gene normalized hair growth in SPF-ASH mice.

TSC2 is crucial for proper hair follicle development and patterning.

Rac1 is essential for proper hair structure and color.

A new genetic variant causes trichothiodystrophy in two brothers, but their mother may carry it without showing symptoms.

A new genetic tool improves the study of hair growth and potential hair disorder treatments.

Lack of functional CYLD in mice leads to early aging and cancer.

Removing REDD1 in mice increases skin fat by making fat cells larger and more numerous.

Alopecia in VDR knockout mice is due to impaired hair cycle initiation, not keratinocyte issues.

RXRα is crucial for hair growth and skin cell function.

The SHJH hr mice with a mutated Hr gene show signs of faster skin aging due to poor antioxidative protection.

Rac1 is crucial for normal hair structure and pigmentation.

Researchers used CRISPR/Cas9 to create a goat with a gene that increased cashmere production by 74.5% without affecting quality.

TCDD changes skin cell growth and keratin production in mice.

New mouse models help study melanocytic cells for melanoma research.

Female mice with disrupted 5α-reductase 1 had significant metabolic issues, including stress response problems, insulin resistance, liver fat buildup, and obesity.

The TCL1 transgenic mouse model is useful for understanding human B-cell leukemia and testing new treatments.

A new mouse model helps study melanocyte cells using GFP expression.