Search

everythinglearnresearchcommunity

for

Search:↓

A new mutation in the XEDAR gene might cause a rare skin condition called hypohidrotic ectodermal dysplasia.

High CRP levels could indicate vitamin D deficiency in people with alopecia areata.

Scientists made a mouse model of a serious skin cancer by changing skin cells with a virus and a specific gene, which is similar to the disease in humans.

The protein folliculin, involved in a rare disease, works with another protein to control how cells stick together and their organization, and changes in this interaction can lead to disease symptoms.

Blocking certain immune signals can reduce skin damage from radiation therapy.

Trichothiodystrophy hair is structurally abnormal with protein and organization issues.

Trichilemmal carcinoma shares genetic traits with other skin cancers, suggesting similar treatment options.

Vitamin D receptor and hairless protein are essential for hair growth.

Researchers found that certain RNA circles in the brain are linked to disease risk, but their exact role in disease is still unknown.

TRPV1 helps regulate hair growth cycles.

FLCN helps control iron levels in cells.

Mutations in the KRT16 gene can cause skin and nail disorders.

The system allows precise control of gene expression in mouse skin, useful for studying skin biology.

A mutation in the vitamin D receptor causes severe resistance to vitamin D, affecting bone health but not hair growth.

Deleting vitamin D and calcium receptors in skin cells increases skin cancer risk by reducing DNA repair and stress response.

Transit-amplifying cells are crucial for tissue repair and can contribute to cancer when they malfunction.

Activated ras can protect kidney cells from a certain substance that causes cell death.

As skin cells mature, vitamin D receptor levels decrease while retinoid X receptor α levels increase.

A new genetic mutation in the ODC1 gene causes developmental delay and other symptoms in a young girl.

A specific genetic trait in tumor cells is linked to longer survival without disease in certain lymphoma patients.

A new gene mutation causes vitamin D-resistant rickets and hair loss in two siblings.

CLE40 and CRN/CLV2 pathways have opposite effects on root growth in Arabidopsis.

KRTAP2-3 could help predict cancer recurrence by identifying specific cancer cells.

LGR5 mainly stays inside cells, moving to the trans-Golgi network, and this process is important for its role in cell signaling.

Targeted protein degraders show promise in treating cancer but need to target more diverse proteins.

The Trichodysplasia spinulosa virus protein can cause abnormal hair growth in mice.

The vitamin D receptor is involved in multiple body functions beyond calcium regulation, including immune response and rapid reactions not related to gene activity.

A protein called PCBP2 controls the production of a hair growth protein by interacting with its genetic message and is linked to hair loss when this control is disrupted.