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Certain gene variations in the Vitamin D receptor may increase the risk of chronic hair loss.

Monilethrix causes brittle hair and hair loss, and it runs in families.

The most different genetic segment between Africans and East Asians is the EDA2R/AR region, with two main types influenced by population changes and natural selection, and linked to baldness.

A new CDH3 gene mutation was found in a Spanish patient with sparse hair and eye issues.

The skin basement membrane is specialized for different tissue interactions, important for hair growth and attachment.

No clear link between specific gene and hair loss in Mexican brothers.

Nε-(carboxymethyl) lysine delays hair growth by blocking a key protein.

Mutations in the KLHL24 gene cause a skin disorder in some Russian families.

miR-199a-3p controls hair growth and is linked to alopecia areata.

A new method can detect mutations in mice by observing changes in hair follicle cells.

A specific gene mutation causes severe skin and nail issues and hair loss.

Telogen effluvium causes temporary hair loss, especially in women.

HLD10 can include increased body hair and Mongolian spots.

Deleting Smad4 and PTEN genes in mice causes rapid, invasive stomach cancer.

Hair loss from telogen effluvium happens when a disturbance or imbalance causes hair to fall out prematurely.

Loss of Dnmt3a and Dnmt3b leads to more aggressive skin tumors, but blocking PPAR-γ can reduce this effect.

Hair follicles in androgenetic alopecia age faster, especially in the front.

A new genetic change in the DSC3 gene is linked to a rare condition causing hair loss and skin blisters in a child.

Losing Memo protein shortens lifespan and affects health.

Scientists are still unsure what triggers the immune system to attack hair follicles in Alopecia areata.

Pachyonychia congenita is linked to a keratin gene on chromosome 17.

A genetic marker linked to a type of hair loss was found in most patients studied.

miR‑339‑5p can slow down hair follicle stem cell differentiation by targeting DLX5.

Melanocyte pathology requires keratinocyte hyperplasia and regulation dysfunction.

Matrical tumors share a common growth mechanism involving the Wnt pathway and consistent PHLDA1 expression.

Human hair keratins K85 and K35 create unique filament patterns important for early hair formation.

Loss of the p53 gene alone causes tumors, and losing both p53 and Rb genes speeds up aggressive skin cancer.

Some people with congenital triangular alopecia have a central tuft of hair in the bald patch, but the cause is unknown.

Newly designed proteins can effectively degrade specific proteins in cells, offering a potential new therapy method.

UTX is crucial for skin differentiation and health, especially in females.