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The system allows precise control of gene expression in mouse skin, useful for studying skin biology.

Scientists made a mouse that can be made to lose hair and then grow it back.

Activating TERT in mice skin boosts hair growth by waking up hair follicle stem cells.

Increased ODC expression makes normally tumor-resistant mice more prone to tumor development.

The study concluded that specific proteins are necessary to maintain the structure that holds epithelial cells tightly together.

miR-29 is a key factor that accelerates aging.

Inhibiting AP-1 changes skin tumor types and affects tumor cell identity.

Inhibiting AP-1 changes skin tumor types and affects tumor cell identity.

Whn is essential for hair growth, and its malfunction causes hair loss.

Mutant mice help researchers understand hair growth and related genetic factors.

AP-1 controls tumor cell type by affecting key signaling pathways.

Blocking Wnt signaling in young mice causes thymus shrinkage and cell loss, but recovery is possible when the block is removed.

miR-22, a type of microRNA, controls hair growth and its overproduction can cause hair loss, while its absence can speed up hair growth.

The study found stem cells in minor salivary glands that can differentiate and are involved in tumor formation when exposed to tobacco.

SOX11 and SOX4 help skin cells act like embryonic cells to heal wounds in mice.

Damaging mitochondrial DNA in mice speeds up aging due to increased reactive oxygen species, not through the p53/p21 pathway.

More dermal papilla cells in hair follicles lead to larger, healthier hair, while fewer cells cause hair thinning and loss.

Adam10 enzyme is crucial for healthy skin and proper Notch signaling.

Reducing Tyrosinase prevents mature color pigment cells from forming in mouse hair.

A protein called desmoglein 3 is important for keeping hair follicle stem cells inactive and helps in their regeneration.

ERG increases SOX9, promoting prostate cancer growth and invasion.

BMI1 is essential for preventing hair greying and maintaining hair color.

Scientists found cells in hair that are key for growth and color.

Overexpressing the mineralocorticoid receptor in mouse skin causes skin thinning, early skin barrier development, eye issues, and hair loss.

Sweat gland stem cells help maintain glands, aid wound healing, and can regenerate skin structures.

Acne is a common skin condition linked to diet, hormones, and genetics, and early treatment can prevent scarring.

Certain mature cells in mouse lungs can turn back into stem cells to aid in tissue repair.

Overexpression of LRIG3 in skin causes hair loss.

Too much β-catenin activity can mess up the development of mammary glands and make them more like hair follicles.

Sweat gland development involves two unique skin cell programs and a temporary skin environment.