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The white wax scale insect's genome shows that complete metamorphosis evolved earlier than thought and highlights differences in male and female development.

Plasmalogens activate a channel in cells that may stimulate hair growth.

TTD patients don't have a higher skin cancer risk because their main issue is with transcription, not DNA repair.

The document concludes that pig iPSCs show promise for transplant therapies and the field is advancing in controlling cell behavior for biology and medicine.

Three genes linked to the development of trichilemmal cysts were found.

MPA increases cancer spread by boosting Eph A2 activity.

Researchers identified new cell types and genes in early hair follicle development.

The PER3 rs772027021 SNP may cause mild skin pigmentation changes in a new subtype of dyschromatosis universalis hereditaria.

Too much keratin 16 in mice skin causes abnormal skin thickening and structure.

Certain genetic markers linked to reproductive potential were identified by their impact on a protein's ability to bind to genes.

Modified β-catenin causes different effects in hair and skin cells, leading to cysts or tumors.

Researchers found a way to turn skin cells into cells that can grow new hair.

The trichohyalin gene is located at chromosomal region 1q21 with other skin-related protein genes.

Long-read RNA sequencing can identify complex gene changes in IFAP syndrome.

Scientists have found a way to grow hair follicles from human cells in a lab, which could help treat hair loss and skin damage.

Hair follicle stem cells need all reprogramming factors to become pluripotent.

SV40 T antigen in hair follicles causes abnormal hair and health issues in mice.

MAD2B slows down the growth of skin cells that are important for hair development by interacting with TCF4.

Loss of the p53 gene alone causes tumors, and losing both p53 and Rb genes speeds up aggressive skin cancer.

The conclusion is that tritium-labeled testosterone metabolites can be made and are better converted into dihydrotestosterone in skin cells than in prostate tissue.

Both single-spin and double-spin PRP methods effectively treat hair loss, with single-spin slightly increasing platelet count more.

Genetically modifying a bacteria and changing its growth conditions significantly increased the production of a chemical called dipicolinic acid.

Using SNP array testing helped quickly find the gene causing Woodhouse-Sakati syndrome in two related individuals.

New method finds genetic links between Type 2 Diabetes and Prostate Cancer not seen before.

New research suggests that skin cell renewal may not require a special type of cell previously thought to be essential.

New RIPK4 gene mutations were found to cause a type of skin and limb birth defect.

Most hair follicle stem cells do not protect their DNA by dividing it unevenly.

Researchers made stem cells from human hair follicle cells with better efficiency than from skin cells.

Loss of the p53 gene alone causes tumors, and losing both p53 and Rb genes speeds up aggressive skin cancer.