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Special immune cells called Tregs can help prevent lung scarring by blocking a specific growth factor.

Type 1/17 inflammation in psoriasis increases skin cell growth due to a molecule that could be a new treatment target.

Blocking certain proteins on immune cells may help treat alopecia areata.

Mice with human fetal thymic tissue and stem cells developed symptoms similar to chronic graft-versus-host disease.

IL-17-producing γδ T cells help improve bone healing.

New findings suggest targeting IL-23 could treat psoriasis, skin cells can adapt to new roles, direct conversion of skin cells to blood cells may aid cell therapy, removing certain tumor cells could boost cancer immunotherapy, and melanoma may have many tumorigenic cells, not just cancer stem cells.

Chronic refractory alopecia areata has more skin-resident memory T cells, and JAK inhibitors may help reduce them.

Hair follicles help attract immune cells to minor skin injuries.

NKG2D+CD4+ T cells are higher in alopecia areata patients and may be involved in the disease.

A protein called lfTSLP is important in causing allergic and other skin diseases and could be a target for treatment.

Treg dysfunction is linked to various autoimmune skin diseases, and understanding Treg properties is key for new treatments.

Thymic transplantation normalized some T-cells but not others, maintaining immune function.

Malt1 protease is essential for regulatory T cell function and could be targeted to boost antitumor immunity.

Specific immune cells cause alopecia areata and blocking certain proteins can prevent it.

Low CD4 counts in HIV patients are linked to more skin disorders.

Higher CD8+ T cell levels are linked to Alopecia areata in Iraqi patients.

Researchers found a specific immune receptor in patients that causes severe skin reactions to a drug.

Recombinant thymosin β4 promotes blood vessel growth and reduces muscle damage.

Interferon, especially alfa interferon, is an effective treatment for cutaneous T-cell lymphoma with manageable side effects.

Neurotrophin-4 increases calcium current in specific mouse neurons through the PI3K pathway.

γδ T cells adapt uniquely to different tissues in mice.

Patients with Alopecia areata have fewer specific immune cells that normally regulate the immune system, which may contribute to the condition.

Scientists found a new type of skin cell that could help with skin repair and these cells work better with a certain protein.

Thymic stromal lymphopoietin (TSLP) promotes hair growth by stimulating specific skin cells.

The CD44-CD49d complex boosts T cell activation and survival in autoimmune disease.

Thymosin beta 4 protects cells from damage by blocking a harmful microRNA and boosting a protective gene.

Thymosin beta-4 promotes hair growth in mice.

CD8+ virtual memory T cells may cause hair loss in alopecia areata.

Activating TLR9 helps heal large wounds and regrow hair by involving a specific type of immune cell.

Hair follicles help skin immune recovery after UVB exposure.