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Patients with certain FoxN1 gene mutations have severe immune issues but normal skin and hair.

Activating TLR9 helps heal large wounds and regrow hair by involving a specific type of immune cell.

FOXN1 gene variants cause low T cells and immune issues from birth.

IL-26 has antimicrobial and pro-inflammatory effects in the chronic skin condition Hidradenitis Suppurativa.

Vδ1+ T-cells in the skin contribute to hair loss in alopecia areata and could be targeted for treatment.

Resident memory T cells and necroptosis may drive fibrosis in eosinophilic fasciitis and morphea.

IL-17-producing γδ T cells help improve bone healing.

PlncRNA-1 helps hair follicle stem cells grow and develop by controlling a specific cell signaling pathway.

Thymus transplantation successfully restored immune function in infants with FOXN1 deficiency.

A 589 nm laser increases IL-2 and IFN-y gene expression in human T-cells.

Innate lymphoid cells type 1 may contribute to alopecia areata by damaging hair follicles.

Nuclear Factor I-C is important for controlling hair growth by affecting the TGF-β1 pathway.

Targeting specific T cells may help treat alopecia areata.

CD8+ T cells play a key role in graft-versus-host disease in certain mice models.

Blocking certain proteins can reduce skin inflammation caused by cancer treatment.

Tcf3 helps cells move and heal wounds by controlling lipocalin 2.

Hair follicle stem cells help heal wounds by turning certain immune cells into ones that reduce inflammation.

Thymic transplantation normalized some T-cells but not others, maintaining immune function.

Memory T cells in the skin balance staying put and moving into the blood, clustering around hair follicles, and increasing in number after infection.

In alopecia areata, certain immune cells increase and express a protein linked to immune activation.

Minor skin injuries can boost hair growth by increasing TNF-α.

Tissue environment greatly affects the unique epigenetic makeup of regulatory T cells, which could impact autoimmune disease treatment.

TGF-β signaling is essential for hair follicle regeneration during wound healing.

CTHRC1 is essential for healing and preventing heart rupture after a heart attack.

Thymus-derived Tregs, not peripherally-derived Tregs, primarily regulate type 1 diabetes in the NOD mouse model.

JMJD3 and NF-κB activate Notch1, which is essential for skin cell movement and wound healing.

Some thymic peptides can increase human hair growth, while others may inhibit it.

TGF-β1 and TNF-α contribute to lung damage after radiation.

Hair follicles may help teach the immune system to tolerate new self-antigens, but this can sometimes cause hair loss.

Targeting thyroid hormone receptor α in liver cells may help treat liver fibrosis.