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Genetically repaired stem cells may treat certain genetic diseases, Th17 cells are key in fighting systemic fungal infections, hair loss in AGA is due to progenitor cell loss, and α-synuclein transfer might contribute to Parkinson's disease progression.

Tph cells are linked to the severity of systemic lupus erythematosus.

IL-18 signaling helps mature Tregs move into the thymus.

IL-18 signaling helps mature Tregs move into the thymus.

Targeting THY1 can improve skin repair and healing.

HTLV-1-associated lichenoid dermatitis (HALD) is linked to an immune response against HTLV-1-infected cells.

People with atopic dermatitis have more CD4+ T cells that respond to a certain bacterial lipid, which may play a role in the skin condition's inflammation.

TLR3 signaling enhances the immunosuppressive properties of human periodontal ligament stem cells.

Certain immune cells in atopic dermatitis skin could be targeted for treatment.

Hair follicle cells help protect against immune attacks by regulating T-cell activity.

Regulatory T cells help prevent autoimmunity and have potential for treating autoimmune diseases.

Hair follicles may help teach the immune system to tolerate new self-antigens, but this can sometimes cause hair loss.

PDE inhibitors, especially PDE4 inhibitors like apremilast, are effective for certain inflammatory skin conditions but have side effects and can be costly.

Higher PD-1 levels are linked to fewer immune cells in hair follicles in alopecia areata.

Vδ1+ T-cells in the skin contribute to hair loss in alopecia areata and could be targeted for treatment.

The CD4 protein may play a role in the behavior of certain skin cells, affecting their growth, movement, and differentiation.

IL-15 is key for T cell function and could help improve treatments for immune-related diseases.

Enhancing Tregs can protect against alopecia areata.

LHTric-1 is a specific antibody useful for studying hair and nail formation.

Plakophilin 1 helps control skin cell immune responses to prevent excessive inflammation.

IL-17 plays a key role in severe hair loss in chronic alopecia areata.

Targeting Vδ1+T-cells may help treat alopecia areata.

Increased regulatory T cell activity may lead to better outcomes in acute diffuse and total alopecia.

RANKL improves the immune response against herpes simplex virus by enhancing T cell activation and could help develop better treatments or vaccines.

Autoimmune reactions may cause both alopecia areata and HAM.

TH antibodies in vitiligo and AA patients recognize the same protein parts.