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UVB exposure in human skin causes macrophages to produce more IL-10 and less IL-12, leading to immunosuppression.

A new vaccine using a porous scaffold boosts immunity and protects against the flu better than traditional methods.

Hair follicles may help teach the immune system to tolerate new self-antigens, but this can sometimes cause hair loss.

The study concludes that as skin matures from infancy to childhood, there are major changes in cell differentiation, stemness, and growth, leading to a stronger skin barrier in older children.

Mice with human fetal thymic tissue and stem cells developed symptoms similar to chronic graft-versus-host disease.

Human body's immune cells are more common in the layer of fat just beneath the skin than in deeper fat layers.

CD8+ T cells expand significantly in alopecia areata, suggesting new treatment targets.

Only one of the two K16 genes on chromosome 17 makes a functional protein for keratin filaments.

Collagen XVII is crucial for skin cell growth and nail health.

Removing certain immune cells in mice causes their hair to enter the growth phase earlier than usual.

Hair follicles are normally protected from the immune system, but when this protection fails, it can cause hair loss in alopecia areata.

Alopecia areata involves complex immune responses, suggesting broader treatments could help.

Hairless mice are more vulnerable to Listeria infection, but gut microbiota can help reduce damage.

Restoring hair follicle immune privilege may help treat alopecia areata.

Patients with thyroid disorders show different symptoms and antibody levels.

Alopecia areata patients have higher levels of certain immune receptors, suggesting new treatment possibilities.

Higher PD-1 levels are linked to fewer immune cells in hair follicles in alopecia areata.

Inhibiting IL-17 and IL-23 improves wound healing in obese, diabetic mice by promoting healing macrophages.

Collagen XVII is crucial for preventing hair and pigmentation loss by maintaining melanocyte stem cells.

TRIV-509 quickly improves skin barrier and cell health in atopic dermatitis.

Targeting THY1 can improve skin repair and healing.

Hair follicles could help develop eye treatments by studying immune responses.

A new mutation in the STING protein causes a range of symptoms and its severity may be affected by other genetic variations; treatment with a specific inhibitor showed improvement in one patient.

People with Alopecia areata have higher levels of certain T regulatory cells in their blood.

Collagen XVII is important for skin aging and wound healing.

Certain immune cells, when activated by specific signals, can encourage hair growth.

Inflammation helps stem cells repair tissue by directing their behavior.

Thymic transplantation normalized some T-cells but not others, maintaining immune function.

BST2 is highly expressed in certain immune cells in alopecia areata, suggesting a role in the disease.

The new vaccine platform led to a stronger immune response and better protection against the flu than the traditional vaccine.