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Newborns with the common rash Erythema Toxicum have many active mast cells in their skin, but these cells don't produce the LL-37 peptide.

Scientists made a mouse model of a serious skin cancer by changing skin cells with a virus and a specific gene, which is similar to the disease in humans.

ILC1-like cells can cause alopecia areata by attacking hair follicles.

IL-19 and IL-24 help cells respond to DNA damage and could be targeted for cancer and age-related disease treatments.

Blocking TSLP reduces skin inflammation and cell overgrowth in psoriasis.

Plasmacytoid dendritic cells, which overproduce IFN-α, may play a crucial role in starting alopecia areata, an autoimmune disease causing hair loss.

The L412F variant of TLR3 is linked to skin infections, more viral infections, and autoimmune issues.

ILC1-like cells can cause alopecia areata by disrupting hair follicle immunity, suggesting a new treatment approach.

Overexpressing Merkel cell virus proteins in human hair follicles can create clusters of cells that resemble Merkel cell cancer.

Lymphocytes, a type of immune cell, are crucial for wound healing as they help remodel damaged areas and reduce inflammation.

Enhancing regulatory T cells may help treat autoimmune diseases like alopecia areata.

Type 1/17 inflammation in psoriasis increases skin cell growth due to a molecule that could be a new treatment target.

Injecting recipient splenocytes into donors' thymus can prevent graft-versus-host disease.

The CD4 protein may play a role in the behavior of certain skin cells, affecting their growth, movement, and differentiation.

BTNL proteins help control inflammatory bowel disease by maintaining specific immune cells.

Targeting Keratin 17 may help overcome cancer therapy resistance.

A new mutation in the STING protein causes a disease with lupus-like symptoms and responds well to a specific inhibitor treatment.

The muscle damage was caused by T-cell large granular lymphocytic leukemia, not dermatomyositis.

Certain immune system genes are linked to a higher risk of psoriasis and psoriatic arthritis, while others may offer protection.

BST2 protein and certain T cells increase in early alopecia areata.

Scientists found a new type of skin cell that could help with skin repair and these cells work better with a certain protein.

Higher levels of certain immune cells in hair follicles may contribute to alopecia areata.

Skin stem cells help create protective immune cells during wound healing.

Targeting specific T cells may help treat alopecia areata.

CD8+ T cells are involved in alopecia areata and may cause disease relapse.

Immune cells might contribute to hair loss caused by a specific mutation.

Keratin 17 is crucial for cell growth in wound healing by aiding protein synthesis.

γδTregs may help treat autoimmune diseases like alopecia areata by promoting hair regrowth and reducing immune attacks.

The hamster polyomavirus middle T antigen is linked to tumors in hamsters and associates with a specific tyrosine kinase.