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Cell-based therapy using specific immune cells may help treat alopecia areata by promoting hair regrowth.

Atopy and altered T cell functions contribute to Alopecia Areata.

Blocking CD44 can reduce leukocyte migration in autoimmune skin diseases.

M-CSF-stimulated myeloid cells can cause alopecia areata in mice.

Folliculotropic mycosis fungoides has unique molecular features and cell interactions that could guide targeted therapy.

TRF1 is crucial for creating and maintaining stem cells and marks both pluripotent and adult stem cells.

Thymic transplantation normalized some T-cells but not others, maintaining immune function.

ILC1-like cells can cause alopecia areata by attacking hair follicles.

Cutaneous lupus patients have higher levels of certain immune cells in their blood and skin.

Innate lymphoid cells type 1 may contribute to alopecia areata by damaging hair follicles.

New research suggests that skin cell renewal may not require a special type of cell previously thought to be essential.

Tcf3 and Lef1 are key in deciding skin stem cell roles.

Certain immune cells in the skin can stop hair from growing.

Human dermal γδT-cells respond to stress in hair follicles, contributing to hair loss.

People with Alopecia areata have higher levels of certain T regulatory cells in their blood.

Autoimmune reactions may cause both alopecia areata and HAM.

Hair follicle stem cells can become corneal-like cells with the help of pax6.

TNC+ fibroblasts play a key role in skin inflammation by interacting with T cells.

IL-1 promotes fat cell growth in skin, while WNT inhibits it and encourages scar formation.

The FOXN1 gene is crucial for developing immune cells and preventing immune disorders.

IL-13 protein is much higher in the skin of atopic dermatitis patients than in healthy skin.

ILC1-like cells can cause alopecia areata by disrupting hair follicle immunity, suggesting a new treatment approach.

Researchers developed a 3D skin model with its own immune and blood vessel cells to better understand skin health and disease.

Keratin 17 is crucial for cell growth in wound healing by aiding protein synthesis.

Certain proteins help nerve cells branch, and other findings relate to cancer, stem cell behavior, and cell division.

HPV16 oncogenes disrupt the normal activity of hair follicle stem cells.

A virus protein can activate a pathway that may lead to abnormal hair follicle development.

People with acne have more CD4+ immune cells in their skin than healthy people.

CD44 variant changes start alopecia areata, but don't maintain it.