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Genetically repaired stem cells may treat certain genetic diseases, Th17 cells are key in fighting systemic fungal infections, hair loss in AGA is due to progenitor cell loss, and α-synuclein transfer might contribute to Parkinson's disease progression.

Botulinum toxin type A significantly reduces scalp psoriasis severity compared to placebo.

Stem cell activity influences autoimmune disease outcomes by affecting immune responses and tissue regeneration.

IL-18 signaling helps mature Tregs move into the thymus.

IL-18 signaling helps mature Tregs move into the thymus.

IL-1 family cytokines are crucial for skin defense and healing, but their imbalance can cause skin diseases.

TNF alpha in skin cells causes weight loss, hair and fat issues, and skin inflammation in mice.

Certain immune system proteins are important for skin healing but can cause problems if there are too many of them.

Memory T cells in the skin balance staying put and moving into the blood, clustering around hair follicles, and increasing in number after infection.

CD8+ T cells play a key role in graft-versus-host disease in certain mice models.

Certain immune cells in atopic dermatitis skin could be targeted for treatment.

TSLP affects atopic dermatitis by increasing sebum and reducing fat through IL-4/IL-13 signaling.

Hair follicle-derived IL-7 and IL-15 are crucial for maintaining skin-resident memory T cells and could be targeted for treating skin diseases and lymphoma.

T cells affect skin cell genes in inflammatory diseases, and therapy can normalize these changes.

AD-derived keratinocytes effectively mimic inflammation in atopic dermatitis.

Tph cells are linked to the severity of systemic lupus erythematosus.

Psoriasis may be chronic because it lacks certain immune system controls that prevent overreaction.

Certain cytokines may cause or be affected by immune skin diseases, suggesting possible new treatments.

CXCL12+ fibroblasts help recruit neutrophils to fight skin infections.

Genes affect cytokine production, which can influence chronic diseases, and certain interventions may help prevent related molecular damage.

IL-15 is key for T cell function and could help improve treatments for immune-related diseases.

IL-18 levels are higher in hidradenitis suppurativa patients and may indicate disease severity.

Recombinant cytokine therapy can cause skin reactions ranging from mild to severe.

Hair follicles help attract immune cells to minor skin injuries.

The CD4 protein may play a role in the behavior of certain skin cells, affecting their growth, movement, and differentiation.

UVB exposure in human skin causes macrophages to produce more IL-10 and less IL-12, leading to immunosuppression.

Type 2 cytokines IL-4 and IL-13 increase skin's vulnerability to Staphylococcus aureus, which could be important for treating atopic dermatitis.

Certain proteins and cytokines are key in causing psoriatic skin issues.

Psoriasis involves an imbalance between certain immune cells, and targeting these could help restore skin health.