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Hair follicle-derived IL-7 and IL-15 are crucial for maintaining skin-resident memory T cells and could be targeted for treating skin diseases and lymphoma.

Cells from a skin condition can create new hair follicles and similar growths in mice, and a specific treatment can reduce these effects.

Lymphotoxin-β is crucial for proper skin development in embryos.

Targeting regulatory T cells may help treat age-related diseases.

Researchers found three types of melanocytes in developing mouse skin, each with different genes and locations.

Memory regulatory T cells need IL-7, not IL-2, to stay in peripheral tissues.

Transglutaminase 2 may control sebocyte maturation and lipid metabolism.

Thymic stromal lymphopoietin (TSLP) promotes hair growth, especially after skin injury.

Cells in hair follicles help create fat cells in the skin by releasing a protein called Sonic Hedgehog.

Skin cancer often starts from Lgr5+ progenitor cells.

BST2 protein and certain T cells increase in early alopecia areata.

Telocytes might help with skin repair and regeneration.

Sox9 levels in brain stem cells affect whether they stay as stem cells or become neurons.

Lgr5+ stem cells do not cause skin tumors.

Hanson's thymus extract sped up growth and development in mice over generations.

Thymosin beta 4 protects cells from damage by blocking a harmful microRNA and boosting a protective gene.

Touch dome keratinocytes in adult skin have traits of different skin cell types.

IL-17 plays a key role in severe hair loss in chronic alopecia areata.

Thymosin β4 helps in cell activities, healing, and organ preservation, and treats hair loss and skin injuries.

Dormant melanoma cells in mice interact minimally with memory T cells due to a suppressive tumor environment.

Memory T cells in the skin balance staying put and moving into the blood, clustering around hair follicles, and increasing in number after infection.

A protein called lfTSLP is important in causing allergic and other skin diseases and could be a target for treatment.

CD4 T cells need IFN-γ to cause hair loss in alopecia areata.

Early regulatory T cells are crucial for normal skin pigmentation.

Skin-derived precursors in hair follicles come from different origins but function similarly.

Two siblings had a rare immune disorder caused by a FOXN1 gene mutation.

CRAC channels are crucial for the development and function of specialized immune cells, preventing severe inflammation and autoimmune diseases.

Clonally expanded CD8+ T cells cause alopecia areata.