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The skin produces and responds to thyroid-stimulating hormone, which is controlled by other thyroid-related hormones.

Thyrotropin-releasing hormone may help control skin and hair growth and could aid in treating related disorders.

Thymic mesenchymal cells have unique gene expression that supports their specific functions in the thymus.

Targeting THY1 can improve skin repair and healing.

A mycobacterial protein shares a similar region with a human skin protein, possibly affecting skin diseases.

Cytokeratins 15 and 19 may help track vitiligo treatment progress.

IL-18 signaling helps mature Tregs move into the thymus.

TH antibodies in vitiligo and AA patients recognize the same protein parts.

K15 is a reliable marker for studying stem cells in dog hair follicle tumors.

Thyroid-related genes are active in skin cells and may affect autoimmune conditions.

Putting thymidine dinucleotide on newborn mice's skin can delay and reduce skin cancer.

IL-18 signaling helps mature Tregs move into the thymus.

Thyroxine (T4) may help heal skin wounds by promoting new skin and blood vessel growth.

A protein called lfTSLP is important in causing allergic and other skin diseases and could be a target for treatment.

Replacing defective mesenchymal cells with normal ones fixes thymic growth issues in 22q11.2DS mouse models.

Removing the thymoma improved the patient's alopecia areata, suggesting a possible link between the two.

Foxn1 gene regulation is crucial for thymus development but not for hair growth.

FoxN1 gene is crucial for proper thymus structure and normal skin appearance.

High concentrations of rosemary and thyme extracts can damage thymus tissue and affect immune markers in chick embryos.

Glucocorticoids and thyroid hormones together are essential for fetal fat development.

The enzyme is crucial for skin cell development and can be activated without proteolytic activation.

Lymphotoxin-β is crucial for proper skin development in embryos.

The new antibody, TYHF-1, specifically targets certain hair-related structures.

Skin tumor cells in patients with tuberous sclerosis have higher levels of a protein called cathepsin B.

Removing the thymoma improved the man's autoimmune conditions.

Keratin 15 expression in skin cells is regulated by two mechanisms involving PKC/AP-1 and FOXM1.

A woman's hair loss improved after removing a tumor in her thymus gland, suggesting hair loss can be linked to such tumors even without a specific muscle weakness condition.

Thymic transplantation normalized some T-cells but not others, maintaining immune function.