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Scientists found a new type of skin cell that could help with skin repair and these cells work better with a certain protein.

T cells affect skin cell genes in inflammatory diseases, and therapy can normalize these changes.

A chimeric keratin partially improved skin structure in mice lacking keratin 5, but didn't fully restore normal skin.

Thymic stromal lymphopoietin (TSLP) promotes hair growth, especially after skin injury.

A new DSG4 gene mutation causes hair defects in a young girl.

Estrogen increases blood vessel growth factor production, while testosterone blocks this increase.

Adding collagenase to trypsin improves cell yield and repigmentation in vitiligo treatment.

Tissue-resident memory T cells can protect against infections and cancer but may also contribute to autoimmune diseases.

THA is a rare condition with no significant clinical consequences if thyroid function is normal.

Helminth protein helps wounds heal better by reducing scarring and promoting tissue growth.

Triphenylphosphine oxide can increase oil production and reduce inflammation in skin cells without affecting TRPM5.

Truncated hHb1 keratin may play a role in breast cancer cell transformation.

The ITGB6 gene is important for tissue repair and hair growth, and mutations can lead to enamel defects and other health issues.

The vitamin D receptor can regulate genes and affect hair and hormone levels without its usual activator.

Trypsin slows hair growth and affects color by causing cell death in hair follicles.

Trichocytic cytokeratins are found in hair, nails, tongue, and thymus cells, showing complex regulation in tissue development.

TRPV3 and TRPV6 channels change structure to regulate calcium and heat responses.

The C-terminal tail of AHF/trichohyalin is essential for organizing keratin filaments in keratinocytes.

Vitamin D receptor helps prevent skin tumors.

A specific gene region can control targeted and responsive gene expression in mice, useful for skin disorder treatments.

Vitexin Compound 1 may help reduce skin aging caused by UVA light.

Trypsin slows hair growth and affects color by causing cell death in hair follicles.

Different γδ T cell types have unique roles in causing alopecia areata.

P144® improves hypertrophic scars by reducing size and thickness and increasing elasticity.

Contact immunotherapy can cause vitiligo in patients with autoimmune conditions.

Tyrosine kinase inhibitors show promise in treating some skin diseases but their definitive role in dermatology is still unclear.

SPT6 prevents excessive skin inflammation by blocking a feedback loop.

Two siblings had a rare immune disorder caused by a FOXN1 gene mutation.

A child with trichothiodystrophy also had autoimmune thyroiditis and anemia, which is a new finding.

Cytokine therapy is promising for treating skin conditions like vitiligo and psoriasis.