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Giving immune serum from vaccinated mice to mice without T cells prevents infection and tumor growth.

Using low-dose IL-2 to increase regulatory T cells might be a safe way to treat type 1 diabetes without severe side effects.

A child with trichothiodystrophy also had autoimmune thyroiditis and anemia, which is a new finding.

The study concludes that as skin matures from infancy to childhood, there are major changes in cell differentiation, stemness, and growth, leading to a stronger skin barrier in older children.

Researchers created a mouse with the same mutation as humans with trichothiodystrophy, showing similar symptoms and confirming the condition is due to defects in DNA repair and gene activity.

Activating TLR3 helps improve skin and hair follicle regeneration after wounds.

Thymoma-associated multiorgan autoimmunity can cause liver damage and affects multiple organs, with limited treatment options and a generally poor prognosis.

Stem cell activity influences autoimmune disease outcomes by affecting immune responses and tissue regeneration.

Telocytes might help with skin repair and regeneration.

Tgm2 helps stabilize dying cells and aids fibroblast attachment to the extracellular matrix.

Thymosin beta 4 may help hair growth and could be a treatment for hair issues.

TLR3 signaling enhances the immunosuppressive properties of human periodontal ligament stem cells.

Different γδ T cell types have unique roles in causing alopecia areata.

Retinoic acid changes skin cells to mucosal cells with goblet cells, needing TG2/Gh, Gbx1, and TGF-beta.

The role of γδT-cells in causing alopecia areata remains unclear.

Activating TLR3 may help produce retinoic acid, important for tissue regeneration.

Blocking TSLP reduces skin inflammation and cell overgrowth in psoriasis.

Thymosin Beta 4 helps human dental pulp cells develop into tooth-forming cells, suggesting it could aid dental regeneration.

Targeting CD200 could be a new treatment for rheumatoid arthritis.

Tertiary lymphoid structures are important in immunotherapy and need more research.

Certain CD8+ T cells attack hair follicles in alopecia areata, suggesting they could be targeted for treatment.

Thymosin beta 4 helps hair grow by boosting stem cell activity.

TLR2 is important for hair growth and can be targeted to treat hair loss.

TSC2-/meth cells can cause skin lesions, hair growth, and lung issues, and may be treated with chromatin remodeling agents.

BTNL2 helps protect hair follicles from immune attacks.

Increased Toll-like receptors in blood cells may contribute to alopecia areata and could be a target for new treatments.

Lower GATA3 levels in mice help hair regrow by changing certain immune cells.

Clonally expanded CD8+ T cells cause alopecia areata.

Inhibiting TYK2 can restore hair growth in alopecia areata.

A new mutation in the STING protein causes a disease with lupus-like symptoms and responds well to a specific inhibitor treatment.