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Scientists found a new type of skin cell that could help with skin repair and these cells work better with a certain protein.

Targeting THY1 can improve skin repair and healing.

Activin increases skin tumor formation, skin Tregs help hair growth, lymph-node removal doesn't improve melanoma survival, cells can revert to stem cells in wound healing, and skin bacteria produce peptides that may treat infections.

Super-enhancers control CD25 expression in specific cell types, affecting immune function.

Expanded CD8+ T cells are linked to Alopecia Areata and may cause relapse after treatment.

The role of γδT-cells in causing alopecia areata remains unclear.

Granulation tissue-derived cells can aid wound healing and serve as an alternative source of stem cells for tissue repair.

TSC2-/meth cells can cause skin lesions, hair growth, and lung issues, and may be treated with chromatin remodeling agents.

Telocytes might help with skin repair and regeneration.

IL-17-producing γδ T cells help improve bone healing.

Lower GATA3 levels in mice help hair regrow by changing certain immune cells.

Regulatory T cells enhance bone formation by influencing cell mechanics.

Blocking interferon-gamma might help treat various autoimmune diseases.

Gamma/delta T cells help defend skin against heavy metals.

TLR2 is important for hair growth and can be targeted to treat hair loss.

TH antibodies in vitiligo and AA patients recognize the same protein parts.

A child with trichothiodystrophy also had autoimmune thyroiditis and anemia, which is a new finding.

Melanoma cells lose their ability to form tumors when placed in a zebrafish embryo environment.

A specific type of immune cell plays a key role in causing alopecia areata and could be a target for treatment.

Targeting Vδ1+T-cells may help treat alopecia areata.

CD4 T cells can cause alopecia areata by activating CD8 T cells to attack hair follicles.

B cells can both help and hinder the body's defense against melanoma.

Activating TERT in mice skin boosts hair growth by waking up hair follicle stem cells.

CTHRC1 helps sweat glands recover by rebuilding nearby blood vessels.

Thymosin beta 4 helps hair grow by boosting stem cell activity.

Researchers created a mouse with the same mutation as humans with trichothiodystrophy, showing similar symptoms and confirming the condition is due to defects in DNA repair and gene activity.

The Itpr3 gene causes a specific hair pattern in mice.

Psoriasis involves an imbalance between certain immune cells, and targeting these could help restore skin health.

A new TP63 mutation was found in a baby with EEC syndrome, showing the need for TREC testing to check for immune issues.