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Four patients with a type of rickets and hair loss had different mutations in their vitamin D receptor gene, causing it to not work properly.

Vitamin D helps skin stem cells heal wounds by working with a key skin protein.

Vitamin D Receptor is crucial for normal skin and hair growth.

Mutations in the vitamin D receptor cause hereditary vitamin D-resistant rickets, leading to poor bone health and requiring high calcium doses for treatment.

Vitamin D is crucial for bone health and preventing serious diseases.

Early detection of specific VDR mutations is crucial for effective treatment and better dental outcomes in children with hereditary vitamin D–resistant rickets.

A rare genetic mutation causes resistance to vitamin D, leading to severe rickets and requiring high doses of calcium and vitamin D for treatment.

Three new gene mutations cause rickets and hair loss, treatable with high calcium and calcidol, but hair regrowth is rare.

A vitamin D receptor mutation causes rickets and affects immune responses.

Vitamin D receptor binds similarly to natural and synthetic ligands, affecting gene regulation.

Two rare genetic diseases cause severe rickets in children due to defects in vitamin D metabolism.

Certain gene variations may protect against skin issues and oxidative stress in women with PCOS.

The testes significantly contribute to vitamin D metabolism and may affect male reproductive health and conditions like hair loss.

Vitamin D is important for skin health and can help treat and prevent various skin conditions.

Vitamin D supplements improve hair growth and metabolism in rabbits.

A rare genetic mutation causes resistance to vitamin D, leading to severe rickets and requires high doses of calcium and vitamin D for management.

Low Vitamin D and its receptors might be linked to chronic hair loss in women.

A genetic mutation caused severe rickets and alopecia in an Indian patient, but high-dose calcium and phosphate treatment improved their condition.

New vitamin D3 forms need the vitamin D receptor to reduce fibrosis in human cells.

A Chinese family had a child with a specific gene mutation causing vitamin D-resistant rickets, but the child improved with calcium and low-dose calcitriol.

Stress activates a special function of the Vitamin D receptor with the help of c-Jun, which can also prevent cell death.

People with vitiligo tend to have lower vitamin D levels.

Patients with Discoid Lupus Erythematosus have lower vitamin D and antioxidant levels and higher oxidative stress.

Men with early hair loss have lower vitamin D levels.

Alopecia areata patients have higher IFN-γ and low vitamin D3 levels.

CD19-CAR T cell therapy may help regenerate skin in systemic sclerosis.

VDR gene variations may affect carbohydrate metabolism in young women with hyperandrogenism.

Low vitamin D levels are linked to different types of hair loss.

Low vitamin D might be one of several factors involved in the hair loss condition alopecia areata.

People with alopecia areata have much lower Vitamin-D levels than healthy individuals.