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Blocking EGFR in mice causes hair loss and skin changes.

Transferrin receptor 1 is essential for normal function in the intestines, pancreas, and skin.

Malt1 protease is essential for regulatory T cell function and could be targeted to boost antitumor immunity.

The lanceolate hair-J mutation in mice helps understand human hair disorders like Netherton's syndrome.

A new mouse gene, Keratin 17n, is mainly found in nail tissue and may explain why mice without Keratin 17 don't have nail issues.

Lhx2 is essential for effective Sonic Hedgehog signaling in early retinal development.

The Foxn1(-/-) nude mouse shows disrupted and expanded skin stem cell areas due to high Lhx2 levels.

CD8+ T cells play a key role in graft-versus-host disease in certain mice models.

Disrupting the Hars2 gene in mice causes hearing loss due to mitochondrial problems and hair cell damage.

Altering the keratin 17 gene in mice hair follicles caused temporary hair issues, but changes were minimal and short-lived.

Lack of Ctip2 in skin cells delays wound healing and disrupts hair follicle stem cell markers in mice.

Akt2 protein is essential for normal cell division in early mouse embryos.

Researchers developed a mouse model that tracks hair growth using bioluminescence, improving accuracy in studying hair cycles.

Researchers created a new mouse model, G4, that mimics human PCOS symptoms and links the condition to a specific gene.

Twist2 is essential for proper skin healing and hair growth in developing mice.

Iron deficiency causes hair loss by affecting hair differentiation and cycling.

Activating Tgr5 may help treat hair loss and bone loss.

CXCL7 is essential for muscle repair by aiding early neutrophil infiltration.

Overexpression of hurpin in mice leads to abnormal skin and higher skin cancer risk.

A gene variant causes patched hair loss in mice, similar to alopecia areata in humans.

Mouse models help study genetic skin diseases.

Alopecia in VDR knockout mice is due to impaired hair cycle initiation, not keratinocyte issues.

The antibody 005-C04 blocks prolactin receptors, causing reversible infertility, impaired lactation, and hair regrowth in female mice.

Prdm1 is necessary for early whisker development in mice but not for other hair, and its absence changes nerve and brain patterns related to whiskers.

A new genetic mutation in mice causes permanent hair loss and skin wrinkling.

The SHJH hr mice with a mutated Hr gene show signs of faster skin aging due to poor antioxidative protection.

High levels of ODC and a mutant Ha-ras gene cause tumors in mice.