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The antibody 005-C04 blocks prolactin receptors, causing reversible infertility, impaired lactation, and hair regrowth in female mice.

Lack of TG2 increases fat storage and lowers cell cleanup in skin oil cells.

B6.Cg-Tyr c−2J Hr hr /J mice have a stronger delayed sunburn reaction and are good for UV research.

A specific metabolite, not a receptor protein, triggers the production of certain kidney enzymes, but this process is disrupted in mice with a mutation causing testicular feminization.

Mouse models are essential for studying and improving genetic traits in agriculture.

The E2 protein affects gene activity in hair follicles of mice.

A specific gene segment can make mouse skin cells glow, helping study hair growth and gene effects.

Activating Kras in mouse skin causes excess skin and hair loss.

Rhino mice show significant meibomian gland changes, making them a potential model for studying gland disorders.

Mouse touch-sensitive nerve cells adjust their connections based on competition with other similar cells.

A specific gene variant (rs4833095) is linked to a higher risk of alopecia areata in Koreans.

A mutation in the Sgkl gene causes defective hair growth in mice.

Toll-like receptor 3 helps repair the skin barrier after UV damage.

The study concluded that specific proteins are necessary to maintain the structure that holds epithelial cells tightly together.

Malt1 protease is essential for regulatory T cell function and could be targeted to boost antitumor immunity.

Human cathepsin V can replace mouse cathepsin L to maintain normal skin and hair in mice.

Researchers created a mouse model of a type of rickets that does not cause hair loss.

The harlequin ichthyosis mouse mutation causes thick skin and early death, resembling a human skin disorder.

SV40 T antigen in hair follicles causes abnormal hair and health issues in mice.

Mutations in the hairless gene in mice affect its expression and lead to a range of developmental issues in multiple tissues.

SkQ1 antioxidant improved health and lifespan in mice.

High levels of ODC and a mutant Ha-ras gene cause tumors in mice.

A specific genetic deletion in goats affects cashmere yield and thickness.

The method effectively induces skin cancer in mice for studying tumor development.

A gene mutation in mice causes increased mast cells and disorganized hair follicles in their skin.

Removing certain brain receptors in mice worsens seizure severity and response to treatment during hormone withdrawal.

mrp/plf-mRNA can indicate tumor-promoting effects in skin.