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Lymphoid-specific helicase (Lsh) is crucial for skin growth, change, and healing after injury.

Recombinant human TSG-6 speeds up wound healing in diabetic mice.

Lactoferrin helps mice grow hair by increasing cell growth and hair follicle development.

Stem cell behavior varies with stimuli, and lineage changes can happen without affecting stem cell division.

The 736T>A mutation in the LIPH gene is common in Japanese people with autosomal recessive woolly hair.

Loss of Dnmt3a and Dnmt3b increases aggressive skin tumors by affecting PPAR-γ.

Overexpressing SSAT enzyme reduces prostate tumor growth in mice.

TGF-β1 and TNF-α contribute to lung damage after radiation.

Mutations in the FOXN1 gene cause severe immune issues but don't affect hair and nails.

The hamster polyomavirus middle T antigen is linked to tumors in hamsters and associates with a specific tyrosine kinase.

LED light therapy at 863 nm wavelength can slow down skin tumor growth and reduce inflammation in mice.

Skin grafts on mice can cause an immune response leading to hair loss, useful for studying human hair loss conditions.

Alopecia areata is influenced by genetics and immune system factors, and better understanding could improve treatments.

The hairless gene in Kunming mice is important for hair and skin, and shows genetic variations.

Krt15+ cells in mice can resist radiation, regenerate tissue, and start tumors, suggesting new cancer treatment targets.

GTL1 is needed to control root hair growth and prevent problems when there are too many nutrients.

Mice without the p21 gene can fully regenerate injured ears due to reduced Sdf1 increase and leukocyte recruitment, suggesting new ways to induce tissue regeneration in mammals.

MRL/MpJ mice's skin wounds heal with scars, unlike their ear wounds which can regenerate.

Stress can cause a type of hair loss in mice lacking the CCHCR1 gene.

The DNMT3B -579G>T polymorphism may increase the risk of colorectal cancer.

Loss of Dnmt3a and Dnmt3b leads to more aggressive skin tumors, but blocking PPAR-γ can reduce this effect.

A specific gene segment can make mouse skin cells glow, helping study hair growth and gene effects.

The p75 neurotrophin receptor is important for hair follicle regression by controlling cell death.

Hairless mice have longer hair follicles and abnormal structures during the catagen phase.