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Activating cannabinoid receptor 1 reduces certain keratin levels, potentially aiding psoriasis treatment.

Ritlecitinib reduces alopecia areata symptoms by blocking JAK3/TEC signaling and T-cell activity.

The protein FLCN is involved in cellular cleanup and is regulated by ULK1.

High levels of ERK activity are key for tissue regeneration in spiny mice, and activating ERK can potentially redirect scar-forming healing towards regenerative healing in mammals.

RIPK1 inhibitors may help prevent alopecia areata by reducing immune cell activity.

Researchers found a specific immune receptor in patients that causes severe skin reactions to a drug.

LGR5 mainly stays inside cells, moving to the trans-Golgi network, and this process is important for its role in cell signaling.

Newly designed proteins can effectively degrade specific proteins in cells, offering a promising alternative for targeted protein degradation.

RpoS helps Borrelia burgdorferi survive in hosts and adapt to different environments.

Regulatory T cells are essential for normal and improved wound healing in mice.

DNMT1 helps turn hair follicle stem cells into fat cells by blocking a specific microRNA.

FTY720 helps transplanted fat survive better by reducing immune rejection and improving blood vessel growth.

FLRG and follistatin have different roles in wound healing.

Blocking TYK2 might be a new way to treat hair loss from alopecia areata.

Blocking mTORC1 reduces skin tumor growth in mice.

NFATc1 is crucial for keeping hair follicle stem cells inactive.

The CD44-CD49d complex boosts T cell activation and survival in autoimmune disease.

Blocking a key energy pathway in human hair follicles can trigger stress responses that stop cell growth.

Delta-opioid receptors affect skin cell circadian rhythms, possibly impacting wound healing and cancer.

The Itpr3 gene causes a specific hair pattern in mice.

Regulatory T cells and mesenchymal stem cells work together to prevent immune system overreactions and tissue damage.

New drugs targeting the JAK-STAT pathway show promise for treating inflammatory skin diseases.

RANKL causes lymph nodes to grow by making certain cells multiply.

Ruxolitinib may effectively treat APS-1 by reducing harmful immune responses.

NDRG1 protein helps infantile hemangioma, a common infant tumor, to grow, and its mismanagement by FOXO1 protein plays a big role in causing the tumor.

Disrupting the Flcn gene in mice causes early kidney cysts and tumors, which can be treated with rapamycin.

Faulty Notch signalling may cause hair follicle changes and inflammation in hidradenitis suppurativa.