Search

everythinglearnresearchcommunity

for

Search:↓

Human dermal fibroblasts are the main cells targeted by a virus that can cause a deadly skin cancer, and a certain inhibitor can effectively block this infection.

Hairless protein and putrescine regulate each other, affecting hair growth and skin balance.

LTBP1 is a key regulator in diseases and a potential target for new treatments.

Researchers developed promising agents for prostate cancer imaging, with the best one showing high potential for clinical use.

Transferrin receptor expression increases iron in mouse skin cells without causing damage.

MEF2C is crucial for normal hair cycle progression.

Defective protein folding due to a mutation is key in ANE syndrome.

mTOR may link different pathways in hair follicle tumor formation.

EGFR helps mouse hair follicles stop growing by reducing certain growth regulators.

A TP63 gene mutation causes significant hair loss and mild skin, nail, and tooth abnormalities.

Researchers found key regions in the mouse hairless gene that control its activity in skin and brain cells, affecting hair follicle function.

Krtap11-1 is important for hair strength and structure.

The scant hair in snthr-1Bao mice is likely caused by a deletion affecting the Plcd1 gene.

Activated ras can protect kidney cells from a certain substance that causes cell death.

TAK-279 effectively reduces psoriasis symptoms and is safe.

Adrenomedullin 2 stops cell growth and causes cell death in human hair follicles.

Neurotrophin-4 increases calcium current in specific mouse neurons through the PI3K pathway.

Cells from a skin condition can create new hair follicles and similar growths in mice, and a specific treatment can reduce these effects.

Transferrin receptor 1 is essential for normal function in the intestines, pancreas, and skin.

PTHrP is higher in certain dog tumors and may act as a local growth factor.

Removing both Atr and Trp53 genes in adult mice causes severe tissue damage and death due to DNA damage.

A truncated protein linked to breast cancer may change cell adhesion.

ΔNp63α stops TAp73β from working in skin cancer by blocking its access to specific genes, not by directly interacting with it.

Certain immune cells in the skin release a protein that stops hair growth by keeping hair stem cells inactive.

A Brazilian male with IFAP syndrome has a unique genetic variant causing his condition.

Polycomb Repressive Complex 1 is crucial for keeping stem cells stable and maintaining healthy adult tissues.

Skin tumor cells in patients with tuberous sclerosis have higher levels of a protein called cathepsin B.

KRT72 gene helps form hair.

αMSH may help regulate immune responses in hair follicles and its disruption could lead to hair loss.