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A woman experienced severe side effects from a drug due to a specific genetic variation, suggesting genetic testing could prevent such risks.

Certain HLA class II alleles increase or decrease the risk of alopecia areata.

RXRα is crucial for hair growth and skin cell function.

Keloid scars may form due to changes in skin cell characteristics and specific protein signaling.

A defective gene causes hair loss and taste insensitivity in BTBR mice.

A specific mutation in the TRPV3 gene causes hair follicle cells to develop improperly, leading to hair loss.

Nonantibiotic macrolides show promise for treating various inflammatory skin conditions.

Pregnancies in a woman with the Donohue mutation were managed with genetic testing, resulting in three healthy infants.

As skin cells mature, vitamin D receptor levels decrease while retinoid X receptor α levels increase.

Methylprednisolone treatment helps most alopecia areata patients, but young age, extensive hair loss, and low vitamin D can affect results.

Tigason improved hair growth in a boy with monilethrix without side effects.

Rare ULBP3 gene changes may raise the risk of Alopecia areata, a certain FAS gene deletion could cause a dysfunctional protein in an immune disorder, and having one copy of a specific genetic deletion is okay, but two copies cause sickle cell disease.

Hair loss in certain young mice is linked to a specific gene and can be caused by lack of iron.

Activating liver X receptors can reduce fibrosis by stopping certain immune cells from releasing harmful proteins.

K6/ODC transgenic mice are effective for quickly identifying cancer-causing chemicals.

Certain IL-18 gene variations may increase the risk of alopecia areata.

Scientists found a new type of skin cell that could help with skin repair and these cells work better with a certain protein.

Overexpressing the Tβ4 gene in goats can increase cashmere production.

Steroid-producing capabilities in certain cancers may contribute to treatment resistance.

Misusing topical corticosteroids can cause skin darkening and other side effects.

Two different mutations in the vitamin D receptor gene cause different symptoms and responses to treatment in Lebanese patients with hereditary rickets.

ILC1 cells contribute to hair loss in alopecia areata.

Prostaglandin D2 increases testosterone production in skin cells through a process involving reactive oxygen species, which could be a new target for treating hair loss and other skin conditions driven by testosterone.

Activating certain cells in hair follicles can prevent hair loss caused by cancer treatments.

Activating TERT in mice skin boosts hair growth by waking up hair follicle stem cells.

Monocyte-derived dendritic cells play a key role in UVB-induced skin sensitivity and inflammation.

A patient with granuloma annulare experienced both isotopic and isomorphic responses, with skin lesions responding to steroids but relapsing after stopping treatment.

A patient developed a blister at the injection site after hepatitis C treatment.

Leydig cell tumors can cause high testosterone and symptoms in postmenopausal women, and removing them improves symptoms.

New mutations in the VDR gene cause vitamin D-resistant rickets without hair loss.