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Folliculin deficiency causes problems with cell division and positioning due to disrupted RhoA signaling and interaction with p0071.

Hair thickness differences help diagnose hair loss severity.

PROTACs have become a breakthrough in medicine by effectively targeting and degrading specific proteins to treat diseases.

Temporal triangular alopecia is a non-scarring hair loss in children, often linked to other health conditions.

Dermoscopy shows that diffuse alopecia areata progresses through specific hair growth stages.

A specific mutation in PA-PLA1α causes abnormal hair growth.

The HGCA tool helps identify genes that work together by analyzing their co-expression patterns.

DKK2 and SOSTDC1 together are necessary for the normal timing of the first regression phase in the hair growth cycle.

The "Two-Cell Assemblage" assay is a new, simple method to identify substances that may promote hair growth.

Skin lesions help identify TCM syndromes in alopecia areata.

Nano-PROTACs could improve drug targeting and delivery by using nanotechnology.

Correcting EDA fibronectin organization and YAP translocation can improve wound healing in fibrotic conditions.

Recognizing perinevoid alopecia and concentric regrowth is important for diagnosing and treating alopecia areata.

Dermoscopy effectively distinguishes between acute total hair loss and other types of female hair loss.

A gene called APCDD1, which controls hair growth, is found to be faulty in a type of hair loss called hereditary hypotrichosis simplex.

Hair density and thickness decrease in specific scalp areas in people with androgenetic alopecia.

The mTurq2-Col4a1 mouse model shows that cells can divide while attached to stable basement membranes during development.

Skin organoids can regenerate hair by forming specific cell units with certain signals.

Targeting specific T cells may help treat alopecia areata.

Two new mutations in the CDH3 gene cause hair loss and vision problems in a young girl.

Certain DNA regions in alpacas are linked to fiber diameter.

Congenital triangular alopecia can occur outside the typical fronto-temporal region.

HDAC4 and HDAC7 are crucial for Th17 cell development and could be targeted to treat inflammatory diseases.

CD44 variant changes start alopecia areata, but don't maintain it.

HLD10 can include increased body hair and Mongolian spots.

Inflammatory diets may increase the risk and severity of alopecia areata.

The system can have a stable solution under certain conditions, helping understand hair loss in Alopecia Areata.

A man had rare skin tumors with bone formation and cholesterol deposits.