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Researchers created a cell line to study hair growth and found specific genes affected by dihydrotestosterone.

S100A3 protein is crucial for hair shaft formation in mice.

Keratin 18 helps diagnose and predict cancer progression and affects cancer growth and spread.

PRMT5 inhibitors effectively fight adenoid cystic carcinoma in salivary glands.

Scientists found key proteins and genes that affect skin and hair health, and identified potential new treatments for hair loss, skin disorders, and wound healing.

New mutations in KRT83 and KRT86 are linked to the hair disorder monilethrix.

Overexpressing NSD3 in mice causes breast cancer-like tumors and gland abnormalities.

Claudin expression changes help the skin respond to injury.

The TCL1 transgenic mouse model is useful for understanding human B-cell leukemia and testing new treatments.

PTHrP regulates various normal body functions, including bone development, skin, and muscle function.

High levels of ODC and a mutant Ha-ras gene cause tumors in mice.

MPZL3 protein helps keep sebaceous gland size and cell growth in check.

The balance between androgen receptor and p53 is crucial for sebaceous gland differentiation.

Markers CRABP1, Nestin, and Ephrin B2 are present in skin cancer environments and may influence their development.

Twist1 helps maintain important features of cells crucial for hair growth by working with Tcf4 and β-catenin.

PINK1 is important for controlling gut immune responses linked to early Parkinson's disease.

CD73 may regulate hair growth and could be targeted for hair growth treatments.

Tsc2-deficient stem cells can help understand and treat TSC-related tumors.

Blocking a specific receptor slows down hair loss in mice.

Fatp4 is crucial for healthy skin development and function.

Trichilemmal carcinoma shares genetic changes with other skin cancers, suggesting similar causes and potential treatments.

Trichothiodystrophy is a rare genetic disorder causing hair issues and sometimes linked to DNA repair defects.

The type 3 IP3 receptor is important for controlling hair loss and growth.

Disrupting the Tsc2 gene in certain cells leads to thicker skin, larger hair, and changes in hair growth signaling, which can be partly reversed with specific treatment.

Mice with a mutated Dsg3 gene showed severe symptoms but not the typical blistering of pemphigus vulgaris.

Mice with a Gsdma3 gene mutation have thicker skin and longer hair follicle openings due to increased β-catenin levels.

Type II keratins are uniquely phosphorylated during stress and mitosis, affecting their structure and function.

ULBP3 levels are higher in Tinea capitis patients and may help predict the disease's severity.

The protein called small nuclear ribonucleoprotein polypeptide E is identified as a cause of a type of hair loss without other symptoms.