Search

everythinglearnresearchcommunity

for

Search:↓

The research developed methods to test drugs that could protect and restore hair follicle protection in a hair loss condition.

NCSTN gene mutation causes abnormal skin cell differentiation and more inflammation, contributing to Hidradenitis Suppurativa.

Stat3 influences keratinocyte stem cell behavior, affecting differentiation and migration.

High concentrations of retinoic acid kill hair follicle stem cells, while low concentrations may help treat skin diseases.

Fibroblasts can be turned into melanocytes for potential skin treatments.

The transgene likely activated an oncogene or interrupted a tumor suppressor gene, causing melanoma in mice.

Anabolic steroids and inflammation can disrupt androgen metabolism, potentially causing health issues.

Certain essential oils can activate a human skin receptor, potentially helping with skin disorders.

Introducing the OTC gene improved symptoms in mice with OTC deficiency.

E6/E7 oncogenes in hair follicles cause continuous hair growth by skipping the resting phase.

Extracellular ATP promotes growth, while adenosine inhibits it in Arabidopsis plants.

We need better ways to test and understand SARMs to make safer and more effective treatments.

Translation levels actively determine keratinocyte cell fate.

Dihydrotestosterone (DHT) increases oil production in skin cells by activating mTOR, and mTOR inhibitors can reduce this effect.

BioPlex-11 improves DNA profiling from telogen hair roots in forensic work.

The method effectively induces skin cancer in mice for studying tumor development.

Different oils penetrate hair differently; monounsaturated oils like olive oil penetrate better than polyunsaturated oils.

The LEF-1 binding site enhances gene expression in hair follicles, with other proteins aiding specific regulation.

Retinoic acid can either maintain stem cells or make them specialize, depending on the cell type.

Normal skin cell renewal doesn't need RAR signaling, but vitamin A-related skin thickening does.

INTASYL is a promising, adaptable RNAi technology for treating skin cancers.

TRM21 helps control flavonoid production and root hair growth in Arabidopsis thaliana.

The PML protein helps prevent skin cancer in mice.

The Megsin-Cre transgene is a new tool for genetic manipulation in the skin and upper digestive tract.

Prostate cancer cells can make their own androgens to activate the androgen receptor, and treatments like abiraterone may increase this ability, suggesting new therapies should target the entire steroid-making pathway.

The enzymes Tet2 and Tet3 are important for skin cell development and hair growth.

PRMT5 inhibitors effectively fight adenoid cystic carcinoma in salivary glands.

C-scores can help predict gain-of-function and loss-of-function mutations.

Transglutaminase does not modify human hair.

The hamster polyomavirus middle T antigen is linked to tumors in hamsters and associates with a specific tyrosine kinase.