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Overexpressing NSD3 in mice causes breast cancer-like tumors and gland abnormalities.

Increasing COX-2 in mouse skin causes bigger sebaceous glands and thinner hair, but stopping COX-2 can reverse hair thinning.

Activating TERT in mice skin boosts hair growth by waking up hair follicle stem cells.

The study developed mouse models to help research and treat hair and sweat gland issues.

Researchers successfully transplanted hair follicles in mice, which survived well and helped in wound healing.

Animal models help study psoriasis but have limitations and don't fully mimic the human disease.

Restoring mitochondrial function in mice reversed their skin wrinkling and hair loss.

Researchers created a new mouse model for studying scleroderma.

BMP signaling helps prevent skin tumors by blocking cancer-promoting pathways.

Over-expression of Sp2 can lead to cancer by preventing proper stem cell differentiation.

Blocking a specific protein signal can make hair grow on mouse nipples.

Covering a wound can stop hair growth by promoting scarring, but boosting a process called Wnt signaling can help hair grow back even when the wound is covered.

Keratin mutations may cause scarring alopecia by damaging hair structure.

Hair loss and aging are caused by the breakdown of a key protein in hair stem cells.

Keratin 15 cells from hair follicles help develop and maintain skin tumors in mice.

The hexosamine pathway helps maintain healthy skin by affecting the skin's structure and possibly increasing hair follicle stem cells.

Scientists developed tools to observe hair regeneration in real time and assess skin health, using glowing mice and light-controlled genes.

High levels of ODC and a mutant Ha-ras gene cause tumors in mice.

Increased ODC expression makes normally tumor-resistant mice more prone to tumor development.

Sunscreens with higher SPF and UVA filters offer the best protection against skin aging from UVA rays.

Melanocyte pathology requires keratinocyte hyperplasia and regulation dysfunction.

Overexpression of the HE4 gene in mice causes eye inflammation and cloudiness.

Basal cell carcinomas in mice can start from hair follicle stem cells and other skin cell types, depending on signaling levels.

Taking triterpenoids from Ganoderma lucidum over a long time can help slow down brain aging and improve overall health in mice.

HPV genes and estradiol increase a cancer-related signaling pathway, which may be targeted for cervical cancer treatment.

Early treatment with the right dose of stem cells can reduce lupus symptoms.

Overexpression of the glucocorticoid receptor in mice causes developmental defects similar to ectodermal dysplasia.

Fluorescent proteins in mouse models effectively visualize tumor blood vessel growth.

Activating β-catenin in certain skin cells speeds up hair growth in mice.

LGR5 is a common marker of hair follicle stem cells in different animals and is important for hair growth and regeneration.