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BMP signaling helps prevent skin tumors by blocking cancer-promoting pathways.

Overexpression of COX-2 causes early hair loss in mice, but can be prevented with a COX-2 inhibitor.

Activating TERT in mice skin boosts hair growth by waking up hair follicle stem cells.

Overexpressing GLI-1 in mice skin can cause tumors like human basal cell carcinomas.

Merkel cells stabilize nerve endings in the skin, and they change independently of each other.

Estrogen is important for keeping adult mouse nipple skin healthy by controlling certain cell signals.

Researchers created a new mouse model for studying scleroderma.

Keratin 17 is important for skin development and may help define skin cell types.

Modified β-catenin can cause different effects in mouse skin cells, leading to cysts or tumors depending on the cell type.

Mutant MK6a transgenes in mice cause blistering, hair loss, and potential human alopecia.

Restoring mitochondrial function in mice reversed their skin wrinkling and hair loss.

The system allows precise control of gene expression in mouse skin, useful for studying skin biology.

The UHS promoter is specific to mouse hair follicles.

Scientists developed tools to observe hair regeneration in real time and assess skin health, using glowing mice and light-controlled genes.

Blocking a specific protein signal can make hair grow on mouse nipples.

Suppressing the HGPS mutation may improve symptoms and suggest reversibility.

Over-expression of Sp2 can lead to cancer by preventing proper stem cell differentiation.

A new method helps grow skin stem cells better, which could improve skin grafts for burn victims.

Modified β-catenin causes different effects in hair and skin cells, leading to cysts or tumors.

ErbB2 signaling is crucial for skin cell growth and cancer development in mice.

Smad4 is important for healthy hair follicles because it helps produce a protein needed for hair to stick together and grow.

Overexpressing the mineralocorticoid receptor in mouse skin causes skin thinning, early skin barrier development, eye issues, and hair loss.

K6 gene expression can be controlled and manipulated in mice for studying skin disorders.

Transferrin receptor expression increases iron in mouse skin cells without causing damage.

Human cathepsin V can replace mouse cathepsin L to maintain normal skin and hair in mice.

Adding TERT and BMI1 to certain skin cells can improve their ability to create hair follicles in mice.

CD34+ cells from fat tissue help form hair follicles and blood vessels in skin.

HPV genes in mice improve ear tissue healing by speeding up skin growth and repair.

Overexpressing IGF-1 in mice leads to skin abnormalities and tumors.

The study showed that a specific DNA sequence can control gene expression in hair growth areas of mice.