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In alopecia areata, certain immune cells increase and express a protein linked to immune activation.

Blue-light activation of TrkA improves hair-follicle stem cells' ability to become neurons and glial cells.

Rapamycin and anti-EGFR antibody reduce LAM/TSC cell migration and blood vessel growth in the uterus.

TLR3 activation helps improve skin and hair follicle healing in mice.

Researchers found a specific immune receptor in patients that causes severe skin reactions to a drug.

Activating TRPV1 can boost hair growth by involving neurons, macrophages, and fibroblasts.

A parasite molecule can speed up skin healing and reduce scarring.

Different γδ T cell types have unique roles in causing alopecia areata.

Certain immune cells in the skin release a protein that stops hair growth by keeping hair stem cells inactive.

Krt15+ cells in mice can resist radiation, regenerate tissue, and start tumors, suggesting new cancer treatment targets.

Mice with human fetal thymic tissue and stem cells developed symptoms similar to chronic graft-versus-host disease.

TEC kinases may help cause inflammation in vitiligo and could be targeted for treatment.

Inflammation helps stem cells repair tissue by directing their behavior.

Intestinal intraepithelial lymphocytes are crucial for gut immunity and maintaining the mucosal barrier.

TCF/Lef1 activity is essential for proper skin cell development and renewal.

Lgr5+ stem cells do not cause skin tumors.

Targeting specific T cells may help treat alopecia areata.

TLR2 is important for hair growth and can be targeted to treat hair loss.

The role of γδT-cells in causing alopecia areata remains unclear.

Tgm2 helps stabilize dying cells and aids fibroblast attachment to the extracellular matrix.

Alopecia areata patients have higher levels of certain immune receptors, suggesting new treatment possibilities.

A specific type of immune cell plays a key role in causing alopecia areata and could be a target for treatment.

Toll-like receptor 3 helps repair the skin barrier after UV damage.

A new TP63 mutation was found in a baby with EEC syndrome, showing the need for TREC testing to check for immune issues.

Skin-resident memory T cells may contribute to chronic alopecia areata and baricitinib could be a potential treatment.

People with Alopecia areata have higher levels of certain T regulatory cells in their blood.

Cells that move well may improve hair loss treatments by entering hair follicles.

B cells can both help and hinder the body's defense against melanoma.

ABCG2 protein marks stem-like skin cells in human epidermis.