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The reconstructed skin model from hair follicles functions like human skin in processing chemicals and can be used to test ingredient safety.

Higher cholesterol levels increase aggressive prostate cancer risk.

Male sexual differentiation is regulated independently, while female differentiation occurs in an androgenic environment, affecting conditions like congenital adrenal hyperplasia.

TRPV1 affects sebaceous gland function and could help treat acne.

Blocking TRPV3 may help treat itch in dry skin conditions.

TRPV4 slows hair growth by affecting hair follicle cells.

Certain essential oils can activate a human skin receptor, potentially helping with skin disorders.

The TRPV3 channel structure changes linked to severe itch and hyperkeratosis were identified using cryo-EM.

The TRPV3 ion channel is important for skin and hair health and could be a target for treating skin conditions.

Acid can block TRPV3 from outside the cell but boost its function from inside.

TRPS1 is crucial for bone, kidney, and hair follicle development.

Activating TRPV1 can boost hair growth by involving neurons, macrophages, and fibroblasts.

Certain immune cells in atopic dermatitis skin could be targeted for treatment.

Activating TRPV3 reduces skin oil production and increases inflammation, potentially causing dry skin issues.

TRPV1 helps regulate hair growth cycles.

A small change in the TRPS1 gene leads to a less severe form of a syndrome affecting hair, nose, and finger development.

TRF1 is crucial for creating and maintaining stem cells and marks both pluripotent and adult stem cells.

Triptolide effectively and safely reduces actinic keratosis lesions in mice.

Dermatologists should consider congenital syndromes like TRPS in young adults with early hair loss.

Olmsted syndrome can be inherited as an autosomal recessive trait due to a rare TRPV3 gene mutation.

Carvacrol activates the TRPV3 channel, which may help design new skin treatments.

mrp/plf-mRNA can indicate tumor-promoting effects in skin.

Activating TRPV4 in skin cells helps regrow hair in mice, possibly offering a treatment for hair loss.

The TRPV3 gene mutation affects hair growth by keeping mice in the growth phase longer, which could help treat hair loss.

A new mutation in the TRPS1 gene caused Trichorhinophalangeal syndrome in a 17-year-old, highlighting the need for genetic testing.

The Itpr3 gene causes a specific hair pattern in mice.

TTNPB helps turn stem cells into neural stem cells, improving depression-like behaviors in rats.

Activating TRPA1 reduces scarring and promotes tissue regeneration.

Trps1 is essential for proper hair follicle development.

A position effect on the TRPS1 gene causes excessive hair growth in humans and mice.