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Low Vitamin D receptor levels in breast cancer are linked to worse outcomes and more bone metastases, and could be a marker for prognosis.

MsPG3 protein gathers at root hair tips, aiding growth.

REV7 is crucial for genome stability and cancer treatment, making it a potential target for therapy.

Thrombospondin-1 is crucial for hair follicle regression and reduced blood vessel growth during the catagen phase.

LPA3 signaling in the uterus is crucial for placental formation and fetal development.

The vitamin D receptor is involved in multiple body functions beyond calcium regulation, including immune response and rapid reactions not related to gene activity.

TLR3 signaling enhances the immunosuppressive properties of human periodontal ligament stem cells.

Matriptase-2 helps control iron levels by suppressing hepcidin, and its deficiency can cause iron-deficiency anemia.

The study concluded that a protein important for hair strength is regulated by certain molecular processes and is affected by growth phases.

GRHL3 is important for controlling gene activity in skin cells during different stages of their development.

CaV1.2 helps activate hair follicle stem cells without calcium flux.

A specific hair protein variant increases the spread of breast cancer and is linked to worse survival rates.

p63 and p73 regulate the vitamin D receptor, affecting cancer cell behavior and vitamin D sensitivity.

A compound named ZCZ90 can increase muscle spindle firing, potentially helping treat muscle spasms and hypertension.

Par3 protein is essential for skin cell balance and stability.

High glycine–tyrosine keratin-associated proteins help make hair strong and maintain its shape.

Vitamin D and its receptor may help prevent skin cancer.

New mutations in the VDR gene cause vitamin D-resistant rickets without hair loss.

Higher substance P levels may cause discomfort in hair loss patients.

Targeting PTEN can improve healing in venous leg ulcers.

A truncated protein linked to breast cancer may change cell adhesion.

Hairless protein can block vitamin D activation in skin cells.

The p75 neurotrophin receptor is important for hair follicle regression by controlling cell death.

Trichohyalin-like proteins are essential for the development of skin structures like hair, nails, and feathers.

Plet-1 protein helps hair follicle cells move and stick to tissues.

Blocking a certain signal in the gp130 receptor can improve tissue healing and lessen osteoarthritis symptoms.

Vav2 and Vav3 proteins help control skin stem cell numbers and activity in both healthy and cancerous cells.

Researchers created a mouse with the same mutation as humans with trichothiodystrophy, showing similar symptoms and confirming the condition is due to defects in DNA repair and gene activity.