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Stress activates a special function of the Vitamin D receptor with the help of c-Jun, which can also prevent cell death.

Some prostate cancers have gene changes that may affect treatment with certain drugs.

Sox13 is a marker for early hair follicle development but not essential for skin and hair growth.

LRIG1 protein affects hair growth by regulating skin receptors, leading to hair loss when overexpressed.

IL-15 helps maintain hair growth and protects the immune status of hair follicles.

Impaired autophagy may contribute to alopecia areata.

The mutated hairless gene causes hair loss by acting as a new type of corepressor affecting thyroid hormone receptors.

Peptide-based PROTACs show promise in targeting hard-to-treat proteins, especially for cancer therapy.

The activity of a specific rat enzyme in the prostate and epididymis is highly dependent on the acidity level.

ALKBH5 helps wounds heal faster by stabilizing PELI2 mRNA.

Blocking mTORC1 activity could increase hair pigmentation and potentially reverse greying.

Hormones during pregnancy and lactation keep skin stem cells inactive, preventing hair growth.

c-Maf positive macrophages help form hypertrophic scars by affecting fibroblasts and collagen production.

Targeting mTOR in myeloid cells may help reduce psoriasis symptoms.

Myc changes chromatin in stem cells, causing them to leave their niche.

A new small peptide may help hair growth by activating a specific receptor and should be tested in humans.

Primary care doctors need to monitor JAK and TYK-2 inhibitors carefully for skin conditions.

The L457(3.43)R mutation in the human lutropin receptor causes increased activity and hormone insensitivity, leading to precocious puberty.

PROTACs show promise in cancer treatment by effectively degrading specific harmful proteins.

Super-enhancers control CD25 expression in specific cell types, affecting immune function.

The vitamin D receptor can regulate genes and affect hair and hormone levels without its usual activator.

Overexpressing SSAT enzyme reduces prostate tumor growth in mice.

Increased Toll-like receptors in blood cells may contribute to alopecia areata and could be a target for new treatments.

Increased methylation of the Filip1l gene may contribute to aggressive skin cancer.

TH antibodies in vitiligo and AA patients recognize the same protein parts.

Researchers found a new gene variant linked to a rare bone disease, which doesn't always cause symptoms in carriers.

TGFβ signaling prevents sebaceous gland cells from producing fats.

Statins may help treat alopecia areata by reducing harmful immune interactions.

PDGFA/AKT signaling is important for the growth and maintenance of certain skin fat cells.