Search

everythinglearnresearchcommunity

for

Search:↓

Meis2 is essential for touch sensation and proper nerve connection to touch receptors in certain skin areas of mice.

Mitochondrial problems in diabetic nerve damage might cause pain by lowering the production of certain nerve-related steroids.

BK and Kv4.2 channels help Merkel cells in rat whiskers sense touch.

Different Hairless isoforms affect Vitamin D receptor activity in hair regulation, with one repressing and the other stimulating it.

Vitamin D receptor can control the hairless gene linked to hair loss even without vitamin D.

Dopaminergic neurons in the gut have diverse subtypes with different neurotransmitter contents.

Vitamin D receptor helps prevent skin tumors.

A specific gene mutation causes vitamin D-resistant rickets and hair loss.

Vitamin D receptor may help protect against UV-induced skin cancer.

Mice without active or present vitamin D receptors maintain normal blood sugar control and islet gene expression when calcium levels are normal.

RANKL improves the immune response against herpes simplex virus by enhancing T cell activation and could help develop better treatments or vaccines.

Four patients with a type of rickets and hair loss had different mutations in their vitamin D receptor gene, causing it to not work properly.

Dopaminergic neurons in the gut have distinct subtypes, some releasing both dopamine and acetylcholine.

Inward rectifier potassium channels are important in many body functions and diseases, and could be potential drug targets.

Cromakalim relaxes various blood vessels, while minoxidil sulphate is more selective; they likely act on different potassium channels.

A single amino acid change in the vitamin D receptor can disrupt its function and lead to hair loss.

Agouti protein affects melanocortin receptors through competitive antagonism and receptor down-regulation.

Vitamin D receptor is essential for proper skin healing after injury.

A vitamin D receptor mutation causes rickets and affects immune responses.

14,15-EET may help reduce poststroke pain by affecting certain brain proteins.

Lack of Vitamin D receptor changes skin structure and increases certain immune cells in the skin.

LGR5 mainly stays inside cells, moving to the trans-Golgi network, and this process is important for its role in cell signaling.

PEA reduces pain by increasing neurosteroid synthesis in the spinal cord.

A mutation in the vitamin D receptor causes severe resistance to vitamin D, affecting bone health but not hair growth.

The study found that certain mutations in the vitamin D receptor can cause rickets and potentially affect hair growth.

The vitamin D receptor can act without its usual activating molecule, affecting hair growth and skin cancer, but its full range of actions is not well understood.

Presynaptic inhibition of certain nerve fibers in cats is mainly controlled by GABA and glycine.

Vitamin D and its receptor may help prevent skin cancer.

Researchers found a specific T cell receptor linked to severe drug reactions like Stevens-Johnson syndrome when patients take carbamazepine.