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A parasite-derived molecule speeds up skin healing and affects immune cell behavior without increasing scarring.

Jagged-1 in skin Tregs is crucial for timely wound healing by recruiting specific immune cells.

LED light therapy at 863 nm wavelength can slow down skin tumor growth and reduce inflammation in mice.

Mice without the vitamin D receptor are more prone to UV-induced skin tumors.

IL-15 is key for T cell function and could help improve treatments for immune-related diseases.

CD44 variant changes start alopecia areata, but don't maintain it.

UV light helped human hair transplants survive in mice without broad immunosuppression.

CD80CD86 deficiency causes hair loss by disrupting regulatory T cells.

CD201+ fascia progenitors are essential for wound healing and could be targeted for treating skin conditions.

PINK1 is important for controlling gut immune responses linked to early Parkinson's disease.

Dandruff is linked to changes in the immune system of hair follicles and skin.

The gene therapy showed significant wound healing and was safe for treating severe RDEB.

Vitamin D and its receptor may help prevent skin cancer.

TGM3 is important for skin and hair structure and may help diagnose cancer.

A child with trichothiodystrophy also had autoimmune thyroiditis and anemia, which is a new finding.

Immune cells and plasma proteins are linked to hair loss, suggesting new treatment options.

Skin changes help detect graft-versus-host reaction early after bone marrow transplants.

ZD1839, a cancer drug, can cause mild skin rashes that are treatable without stopping the medication.

Vitamin D3 speeds up skin healing by helping skin stem cells grow and develop.

VDAC2 promotes cell death in cashmere goat hair follicles through the P53 pathway.

The boy's severe skin disorder is caused by two new mutations in his TGM1 gene.

AD-derived keratinocytes effectively mimic inflammation in atopic dermatitis.

Blocking CD44 can reduce leukocyte migration in autoimmune skin diseases.

Trichothiodystrophy is a rare genetic disorder causing hair issues and sometimes linked to DNA repair defects.

CD4 is crucial for maintaining skin stem cell balance and aiding wound healing.

Skin cells control immune cell placement, helping the skin respond better to challenges.

CD133+ cells are crucial for hair growth.

A specific group of early-stage melanocytes is reduced in vitiligo-affected skin, which may explain treatment resistance.

A mutation in the VDR gene affects hair cycling without needing ligand binding.

Cells that move well may improve hair loss treatments by entering hair follicles.