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Intravenous and oral calcium effectively treated vitamin D dependent rickets type II, improving growth and bone health.

Two rare genetic diseases cause severe rickets in children due to defects in vitamin D metabolism.

A boy with severe Vitamin D-resistant rickets did not respond to treatment and lacked a common symptom, suggesting a need for alternative treatments.

Some people have genetic mutations that make them resistant to vitamin D, leading to rickets even with enough vitamin D intake.

A 4-year-old boy with a rare type of rickets and hair loss improved in bone health but not hair growth after vitamin D and calcium treatment.

Child with rickets improved with a specific vitamin D therapy, but alopecia did not change.

Black patients with a specific type of hair loss have a much higher chance of lacking enough vitamin D.

Patients with the same vitamin D receptor mutation showed different symptoms due to other factors.

Vitamin D deficiency is common in people with certain types of hair loss, like alopecia areata and female pattern hair loss.

The authors maintain there is a significant link between vitamin D deficiency and central centrifugal cicatricial alopecia in Black patients, and call for more research on the topic.

A rare genetic mutation causes resistance to vitamin D, leading to severe rickets and requires high doses of calcium and vitamin D for management.

A child with a rare vitamin D-resistant condition improved with treatment.

A rare genetic disease causes rickets and often hair loss in young children, which can improve with specific treatments.

VDDR I and II are genetic disorders affecting vitamin D use, causing rickets, with VDDR I treatable by vitamin D supplements and VDDR II needing high doses and calcium.

Persistent hyperparathyroidism doesn't stop rickets healing or phosphate level normalization in VDDR2A.

A 2-year-old girl with a rare vitamin D disorder had rickets and hair loss, but treatment was ineffective due to poor compliance.

Children with Vitamin D-dependent rickets type II may have severe dental issues that need a team of specialists for proper care.

Calcium supplements improved bone deformities but not skin papules or hair loss.

Vitamin D Dependent Rickets Type-2A can cause complete hair loss and bone growth issues in infants.

Gene therapy helped rats with a specific type of rickets grow hair without severe inflammation.

Growth hormone therapy can improve symptoms and growth in children with Vitamin-D Dependent Rickets Type-2.

A rare genetic mutation causes resistance to vitamin D, leading to severe rickets and requiring high doses of calcium and vitamin D for treatment.

A child with a rare type of rickets showed some improvement with high doses of vitamin D, but such conditions often respond poorly to treatment.

Most female students in Aden, Yemen, have low vitamin D levels, causing health issues.

A child with rickets and hair loss might have a rare type of rickets that doesn't improve much with usual vitamin D and calcium treatment.

Vitamin D receptor can control the hairless gene linked to hair loss even without vitamin D.

Patients with the same genetic mutation for vitamin D-resistant rickets showed different symptoms but all improved with treatment except for hair loss.

Two different mutations in the vitamin D receptor gene cause different symptoms and responses to treatment in Lebanese patients with hereditary rickets.

Deformable vesicular carriers improve drug delivery for skin conditions and systemic treatments.

The testes significantly contribute to vitamin D metabolism and may affect male reproductive health and conditions like hair loss.