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The vitamin D receptor helps control hair growth and may protect against certain skin tumors.

The vitamin D receptor is involved in multiple body functions beyond calcium regulation, including immune response and rapid reactions not related to gene activity.

Two siblings with a genetic mutation had a form of rickets that doesn't respond to vitamin D.

Early detection of specific VDR mutations is crucial for effective treatment and better dental outcomes in children with hereditary vitamin D–resistant rickets.

Certain VDR gene changes can affect melanoma risk.

The vitamin D receptor has many roles in the body beyond managing calcium, affecting the immune system, hair growth, muscles, fat, bone marrow, and cancer cells.

Vitamin D receptor helps control hair growth and could be used to treat certain skin tumors.

Low Vitamin D receptor levels in hair follicles may contribute to alopecia areata.

Vitamin D receptor gene changes don't affect alopecia areata risk.

Vitamin D receptor binds similarly to natural and synthetic ligands, affecting gene regulation.

Mice without active or present vitamin D receptors maintain normal blood sugar control and islet gene expression when calcium levels are normal.

A specific gene mutation causes vitamin D-resistant rickets and hair loss.

A boy with Oculodentodigital syndrome had a unique GJA1 gene mutation causing his symptoms.

Tunisian children with hereditary vitamin D-resistant rickets showed improvement with calcium treatment, and new genetic mutations were identified.

A new gene mutation causes vitamin D-resistant rickets and hair loss in two siblings.

RCVS should be considered in thunderclap headaches, and MRA and DSA are better for diagnosis than CT angiogram.

Four patients with a type of rickets and hair loss had different mutations in their vitamin D receptor gene, causing it to not work properly.

A man had two rare autoimmune diseases that might be connected.

Intravenous calcium therapy is a safe and effective treatment for a rare type of rickets.

A rare genetic mutation causes resistance to vitamin D, leading to severe rickets and requiring high doses of calcium and vitamin D for treatment.

VDC-1101 shows potential as a treatment for canine cutaneous T-cell lymphoma.

A new classification for trichothiodystrophy helps identify genetic causes and potential treatments.

A rare genetic mutation causes resistance to vitamin D, leading to severe rickets and requires high doses of calcium and vitamin D for management.

Vitamin D receptor gene variations are not linked to alopecia areata.

Vitamin D receptor helps prevent skin cancer and supports skin health.

Vitamin D receptor FokI gene variation is not linked to alopecia areata.

The vitamin D receptor is crucial for bone health and affects various body systems, with mutations potentially leading to disease.

DDR-II hair follicle cells don't respond to 1, 25-dihydroxyvitamin D3, aiding diagnosis.

A single amino acid change in the vitamin D receptor can disrupt its function and lead to hair loss.

TTD patients don't have a higher skin cancer risk because their main issue is with transcription, not DNA repair.