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The vitamin D receptor can act without its usual activating molecule, affecting hair growth and skin cancer, but its full range of actions is not well understood.

Mice lacking steroid 5α-reductase 2 show less aggression and better impulse control.

A gene deletion causes the "hairless" trait in Iffa Credo rats.

Vitamin D Receptor is needed for hair growth in mice but not for skin stem cell maintenance.

VEGFR-2 is active in hair follicles, sebaceous glands, sweat glands, and skin on the human scalp.

Ezh2 controls skin development by balancing signals for dermal and epidermal growth.

Low Vitamin D receptor levels in breast cancer are linked to worse outcomes and more bone metastases, and could be a marker for prognosis.

Certain genetic variants in ERN1, TACR3, and SPPL2C are linked to when Alzheimer's disease starts.

PRC2 is essential for maintaining intestinal cell balance and aiding regeneration after damage.

Id proteins, especially Id2 and Id3, are crucial for hair follicle development and stem cell regulation.

Vitamin D receptor is essential for healthy hair growth.

LEF1 interacts with Vitamin D Receptor, affecting hair follicle regeneration and this could be linked to hair loss conditions.

Mice without the vitamin D receptor have bone issues and other health problems, suggesting vitamin D is important for preventing various diseases in humans.

HtrA2 activity is crucial for normal hair growth by regulating fat cell development.

Scientists created a cell model to study and find treatments for a skin disease called RDEB.

CRABP2 helps increase the growth of cells important for hair growth by activating a specific growth pathway.

A new genetic variant causes trichothiodystrophy in two brothers, but their mother may carry it without showing symptoms.

A gene called APCDD1, which controls hair growth, is found to be faulty in a type of hair loss called hereditary hypotrichosis simplex.

The mutant HR bmh protein mis-localizes in cells, affecting skin and hair development.

The gene therapy showed significant wound healing and was safe for treating severe RDEB.

The human type II 5α-reductase gene, linked to certain male health conditions, has a specific structure and low similarity to other related genes.

KRT72 gene helps form hair.

A mutation in the iRhom2 gene causes hairless mice due to abnormal hair follicle development.

VEGFR-2 activation is likely involved in hair follicle growth, survival, and development.

Vitamin D receptor is essential for healthy bones and skin.

The frizzy mouse and hairless rat mutations are due to changes in the Prss8 gene.

Mice without the vitamin D receptor are more prone to UV-induced skin tumors.

A specific gene variant is linked to heart disease, increased heart muscle, curly hair, and thick skin on palms and soles.

A gene mutation causes curly hair and hair loss in rats.