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The vitamin D receptor helps control hair growth and may protect against certain skin tumors.

The mutant HR bmh protein affects hair follicle formation by failing to repress vitamin D receptor activity.

The vitamin D receptor has many roles in the body beyond managing calcium, affecting the immune system, hair growth, muscles, fat, bone marrow, and cancer cells.

Vitamin D receptor is essential for adult hair growth.

Vitamin D is important for skin cell growth and health, and its active form and receptor play key roles in skin and hair processes.

Calcium supplements improved bone deformities but not skin papules or hair loss.

A rare genetic mutation causes resistance to vitamin D, leading to severe rickets and requiring high doses of calcium and vitamin D for treatment.

Certain gene variations may protect against skin issues and oxidative stress in women with PCOS.

A child with a rare vitamin D-resistant condition improved with treatment.

RXRα is crucial for hair growth and skin cell function.

The VDR gene polymorphism does not affect BDNF levels in autoimmune thyroiditis and hypothyroidism patients.

HVDRR is caused by VDR gene mutations, leading to vitamin D resistance, treatable with high calcium doses, but alopecia remains permanent.

Diet can prevent bone issues but not hair loss in mice lacking vitamin D receptors.

Vitamin D and calcium are crucial for proper skin wound healing.

A rare genetic mutation causes resistance to vitamin D, leading to severe rickets and requires high doses of calcium and vitamin D for management.

Disrupting Acvr1b in mice causes severe hair loss and thicker skin.

Removing the p75 gene in mouse skin cells didn't affect their skin or hair growth.

The PP2A-B55α protein is essential for brain and skin development in embryos.

Knocking out certain genes in mice helps understand skin and hair growth problems.

The study created a mouse model to mimic degenerative diseases for testing tissue repair and new therapies.

A new mouse model for vitiligo helps study immune responses and potential treatments.

Knockout mice showed anemia and hair loss, suggesting other ways exist for iron absorption.

The scant hair in snthr-1Bao mice is likely caused by a deletion affecting the Plcd1 gene.

Homozygous K5Cre transgenic mice have wavy hair and faster cancer progression.

PCR genotyping in cre-loxP mice can be inaccurate due to unintended gene deletions in non-target tissues.

Deleting part of a gene in mice causes wavy hair and high pup loss.

Removing certain brain receptors in mice worsens seizure severity and response to treatment during hormone withdrawal.

Gene knockout mice developed scars similar to human hypertrophic scars, useful for studying scar progression.