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Mutations in specific genes cause certain congenital defects in dogs, aiding in understanding similar human diseases.

Old C57BL/6 mice with unsynchronized hair cycles show less melanin in their spleens.

Replacing defective mesenchymal cells with normal ones fixes thymic growth issues in 22q11.2DS mouse models.

Loss of a specific protein in skin cells causes symptoms similar to psoriasis.

Androgen loss may speed up hair greying.

Overexpression of hurpin in mice leads to abnormal skin and higher skin cancer risk.

The type of tumor suppressor affects the form of skin cancer from hair follicle stem cells.

FoxN1 gene is essential for proper thymus structure and preventing hair loss.

Mice can regrow hair on wounds due to specific cell interactions and mechanical forces not seen in rats.

Keratin 16 delays skin maturation and affects skin and hair development in mice.

The Megsin-Cre transgene is a new tool for genetic manipulation in the skin and upper digestive tract.

The mutated hairless gene causes hair loss by acting as a new type of corepressor affecting thyroid hormone receptors.

Copper therapy improved health and enzyme activity in mice with copper deficiency.

The fuzzy gene is crucial for controlling hair growth cycles.

FoxN1 gene is crucial for proper thymus structure and normal skin appearance.

Loss of Dnmt3a and Dnmt3b leads to more aggressive skin tumors, but blocking PPAR-γ can reduce this effect.

The study developed mouse models to help research and treat hair and sweat gland issues.

Neurosteroid withdrawal increases α4 subunit expression in the hippocampus, which may relate to catamenial epilepsy in women.

Mice with extra PKCδ resist chemical-induced skin cancer but not UV-induced.

Gonadal hormones significantly affect the severity of alopecia areata in mice.

Genetic mutations that disrupt homocysteine breakdown lead to increased damage in mouse hair keratin.

The 14-3-3σ gene is essential for preventing hair loss.

Different types of skin on mice have unique chemical profiles related to how they grow and react to stress.

A rare genetic mutation causes severe immune issues, hair loss, and nail problems.

K6 gene expression can be controlled and manipulated in mice for studying skin disorders.

Ablative fractional laser treatment nearly matches the gene reduction effects of topical vismodegib in skin cancer.

MILL molecules are unique immune proteins in mice that don't need TAP to appear on cell surfaces.

Gene regulation evolved differently in mouse and chicken skin, but remained stable in their trunks.

Deleting the Far2 gene in mice causes sebaceous gland issues and patchy hair loss.

TNF alpha in skin cells causes weight loss, hair and fat issues, and skin inflammation in mice.