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Measuring 24-OHase induction helps identify defects in vitamin D processing and predict treatment response.

Vitamin D and its receptor may help prevent skin cancer.

The skin produces and uses vitamin D for bone health, cell growth, and immune function.

New vitamin D3 forms need the vitamin D receptor to reduce fibrosis in human cells.

Vitamin D may help treat hair disorders.

The research showed that Vitamin D and its receptor are important for healthy bones and normal hair and skin in rats.

A rare genetic mutation causes resistance to vitamin D, leading to severe rickets and requires high doses of calcium and vitamin D for management.

Diet and sunlight are important for maintaining good vitamin D levels.

A boy with severe Vitamin D-resistant rickets did not respond to treatment and lacked a common symptom, suggesting a need for alternative treatments.

HVDRR is caused by VDR gene mutations, leading to vitamin D resistance, treatable with high calcium doses, but alopecia remains permanent.

Vitamin D is made by the skin, helps control various body functions, and affects skin health and immunity.

The vitamin D receptor can regulate genes and affect hair and hormone levels without its usual activator.

Vitamin D is important for regulating calcium, phosphate, and hair health.

Vitamin D3 affects cell differentiation in specific skin areas.

Vitamin D receptor may help protect against UV-induced skin cancer.

A rare genetic mutation causes resistance to vitamin D, leading to severe rickets and requiring high doses of calcium and vitamin D for treatment.

Vitamin D3 may protect rat hair follicles from radiation damage.

The vitamin D receptor can affect gene activity even without its usual hormone in hair and skin.

Vitamin D is important for skin cell growth and health, and its active form and receptor play key roles in skin and hair processes.

Vitamin D receptor helps prevent skin tumors.

Mutations in the vitamin D receptor cause hereditary vitamin D-resistant rickets, leading to poor bone health and requiring high calcium doses for treatment.

New mutations in the VDR gene cause vitamin D-resistant rickets without hair loss.

Vitamin D receptor helps prevent skin tumors.

Removing part of the vitamin D receptor stops vitamin D from working properly.

Patients with the same genetic mutation for vitamin D-resistant rickets showed different symptoms but all improved with treatment except for hair loss.

Genetically modified rats help reveal how vitamin D affects bone and skin health.

A genetic mutation caused severe rickets and alopecia in an Indian patient, but high-dose calcium and phosphate treatment improved their condition.

Child with rickets improved with a specific vitamin D therapy, but alopecia did not change.

1α,25-dihydroxyvitamin D3 directly affects cartilage growth and development.