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The vitamin D receptor is essential for skin stem cells to grow, move, and become different cell types needed for skin healing.

Most Hairless gene mutations reduce its ability to work with the Vitamin D Receptor, which might explain a certain type of hair loss.

People with alopecia areata often have lower vitamin D levels, which are linked to more severe and longer-lasting hair loss, but vitamin D receptor levels in the skin don't show the same pattern and don't predict treatment success.

Vitamin D3 may protect rat hair follicles from radiation damage.

Low vitamin D levels are common in people with Alopecia Areata.

Oral vitamin D treatment can help some children with alopecia areata who have vitamin D deficiency.

Low ferritin levels might be linked to telogen effluvium, but vitamin D levels are not.

Skin cells produce and activate vitamin D, which regulates skin functions and supports hair growth.

Vitamin D and its receptor regulate skin functions like cell growth, immunity, hair cycle, and tumor prevention.

Three siblings with a genetic form of rickets showed different symptoms of the disease.

Two different mutations in the vitamin D receptor gene cause different symptoms and responses to treatment in Lebanese patients with hereditary rickets.

Children with autoimmune hair loss have similar vitamin D levels to healthy kids, suggesting no extra screening is needed.

Patients with Discoid Lupus Erythematosus have lower vitamin D and antioxidant levels and higher oxidative stress.

Removing part of the vitamin D receptor stops vitamin D from working properly.

Vitamin D is important for skin health and may affect conditions like psoriasis and hair loss, but more research is needed to understand its role fully.

Topical vitamin D3 does not prevent hair loss from chemotherapy.

Vitamin D3 analogs can promote hair growth in mice genetically prone to hair loss.

The new delivery system improved raloxifene's skin absorption and effectiveness against cancer cells.

Topical vitamin D is useful for some skin conditions but not effective for others, and more research is needed.

Four patients with a type of rickets and hair loss had different mutations in their vitamin D receptor gene, causing it to not work properly.

Low vitamin D receptor levels found in hair loss patients; topical vitamin D treatment suggested.

Vitamin D deficiency is common in women with PCOS and linked to some metabolic problems, but not the main cause of their metabolic issues.

Vitamin B12 deficiency can cause skin darkening and increased blood vessels, but treatment can improve symptoms.

Vitamin D made by the skin plays a role in immune defense and skin health, and more research is needed to understand its full effects.

Low iron levels are a significant risk factor for hair loss, while high vitamin D levels might be a response to hair loss, not a cause.

The method can measure vitamin B3 levels in human hair accurately.

Vitamin D may help reduce antibody levels in men with autoimmune thyroiditis and early-onset androgenic alopecia, but has no significant impact on thyroid function.

Hair loss can be a key sign of a rare type of rickets when vitamin D treatment doesn't work and advanced tests aren't available.

The hair test for vitamin D could be a useful alternative to blood tests, providing a longer-term vitamin D status, but more research is needed.

Rubbing calcipotriol on the skin works better than taking vitamin D pills for treating mild to moderate alopecia areata.