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Vitamin D receptor is crucial for starting hair growth after birth.

Vitamin D receptor is essential for hair growth signaling.

Vitamin D receptor is essential for proper skin healing after injury.

Vitamin D and its receptor may help prevent skin cancer.

Vitamin D receptor is essential for hair growth and preventing hair loss.

Vitamin D receptor may help protect against UV-induced skin cancer.

Vitamin D receptor is essential for healthy hair growth.

A new gene mutation causes vitamin D resistance and rickets, treatable with calcium therapy.

Vitamin D receptor helps control skin cell growth and hair development.

A mutation in the vitamin D receptor causes severe resistance to vitamin D, affecting bone health but not hair growth.

A specific gene mutation causes vitamin D-resistant rickets and hair loss.

The vitamin D receptor is essential for skin stem cells to grow, move, and become different cell types needed for skin healing.

Most Hairless gene mutations reduce its ability to work with the Vitamin D Receptor, which might explain a certain type of hair loss.

The vitamin D receptor can regulate genes and affect hair and hormone levels without its usual activator.

A girl had rickets due to a gene mutation affecting vitamin D response.

LEF1 interacts with Vitamin D Receptor, affecting hair follicle regeneration and this could be linked to hair loss conditions.

The review found no clear link between vitamin D receptor gene variations and polycystic ovary syndrome.

A new gene mutation causes vitamin D resistance and hair loss in two unrelated girls.

The vitamin D receptor can work without its usual activating molecule.

Stress activates a special function of the Vitamin D receptor with the help of c-Jun, which can also prevent cell death.

A Pomeranian dog had rickets due to a new gene mutation, leading to severe symptoms and euthanasia.

People with alopecia areata often have lower vitamin D levels, which are linked to more severe and longer-lasting hair loss, but vitamin D receptor levels in the skin don't show the same pattern and don't predict treatment success.

Vitamin D receptor is crucial for healthy hair growth and preventing hair loss.

Alopecia in VDR knockout mice is due to impaired hair cycle initiation, not keratinocyte issues.

Removing vitamin D and calcium receptors in mice skin cells slows down skin wound healing.

Hairless protein can both repress and activate vitamin D receptor functions, affecting gene regulation.

Vitamin D Receptor is needed for hair growth in mice but not for skin stem cell maintenance.

Four patients with a type of rickets and hair loss had different mutations in their vitamin D receptor gene, causing it to not work properly.

Vitamin D receptor is important for regulating hair growth and wound healing in mice.

Certain mutations in the hairless protein disrupt its ability to regulate the hair cycle.