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Vitamin D made by the skin plays a role in immune defense and skin health, and more research is needed to understand its full effects.

The vitamin D receptor can work without its usual activating molecule.

The vitamin D receptor has many roles in the body beyond managing calcium, affecting the immune system, hair growth, muscles, fat, bone marrow, and cancer cells.

Vitamin D receptor helps prevent skin tumors.

1,25-dihydroxyvitamin D3 helps melanocyte precursors from hair follicles grow and produce melanin, aiding vitiligo treatment understanding.

Vitamin D receptor helps prevent skin cancer and supports skin health.

Four patients with a type of rickets and hair loss had different mutations in their vitamin D receptor gene, causing it to not work properly.

A child with a rare type of rickets showed some improvement with high doses of vitamin D, but such conditions often respond poorly to treatment.

Vitamin D affects many body functions and its interaction with microRNAs could help treat related diseases.

Vitamin D is made by the skin, helps control various body functions, and affects skin health and immunity.

Vitamin D receptor helps prevent skin tumors.

Vitamin D is crucial for bone health and preventing serious diseases.

Vitamin D could be important for many skin functions and synthetic versions may treat various skin diseases.

The vitamin D receptor is crucial for bone health and affects various body systems, with mutations potentially leading to disease.

Mutations in the vitamin D receptor cause hereditary vitamin D-resistant rickets, leading to poor bone health and requiring high calcium doses for treatment.

The vitamin D receptor is involved in multiple body functions beyond calcium regulation, including immune response and rapid reactions not related to gene activity.

Removing part of the vitamin D receptor stops vitamin D from working properly.

Vitamin D3 is crucial for health, preventing issues like bone disorders and hair loss.

Vitamin D receptor actions without binding are crucial for healthy skin and hair.

Vitamin D receptor is essential for preventing hair loss by regulating hair cycle stages.

The skin produces and uses vitamin D for bone health, cell growth, and immune function.

A boy with severe Vitamin D-resistant rickets did not respond to treatment and lacked a common symptom, suggesting a need for alternative treatments.

The vitamin D receptor can affect gene activity even without its usual hormone in hair and skin.

A new gene mutation causes vitamin D resistance and rickets, treatable with calcium therapy.

Skin cells produce and activate vitamin D, which regulates skin functions and supports hair growth.

New mutations in the VDR gene cause vitamin D-resistant rickets without hair loss.

The research showed that Vitamin D and its receptor are important for healthy bones and normal hair and skin in rats.

Vitamin D and its receptor may help prevent skin cancer.

A rare genetic mutation causes resistance to vitamin D, leading to severe rickets and requires high doses of calcium and vitamin D for management.