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The fuzzy gene is crucial for controlling hair growth cycles.

Removing Mediator 1 from certain mouse cells causes teeth to grow hair instead of enamel.

Msx-2 gene removal speeds up skin wound healing in mice.

Overexpression of IKZF1 and Ikaros causes hair loss in mice similar to alopecia areata.

Nut extracts promote hair growth in mice.

Two rare genetic diseases cause severe rickets in children due to defects in vitamin D metabolism.

Mice without collagen VI have slower hair growth normally but faster regrowth after injury.

miRNAs are crucial for hair growth and skin health.

The Trichodysplasia spinulosa virus protein can cause abnormal hair growth in mice.

Acid inside cells speeds up aging and turns on aging signs in mice.

Copper deficiency lowers blood and liver copper levels in rats, but not in hair or bones.

Ganoderma lucidum extract and microneedle therapy promote hair growth in mice.

A mother's western diet can make her milk toxic, causing inflammation and hair loss in babies.

TCDD changes skin cell growth and keratin production in mice.

Dormant melanoma cells in mice interact minimally with memory T cells due to a suppressive tumor environment.

Retinoic acid affects male and female muscle energy use and function differently.

Scientists made a mouse that shows how a specific protein in the skin changes and affects hair growth and shape.

Trps1 is essential for proper hair follicle development.

Mice's intestinal uptake of pantothenic acid is not affected by dietary levels.

Overexpression of PTHrP in chondrocytes causes short-limbed dwarfism and delayed bone formation in mice.

Smad1 and Smad5 are essential for hair follicle development and stem cell sleepiness.

The gene Ascl4 is not necessary for the development of hair, teeth, or mammary glands.

The retinoid receptor antagonist effectively disrupts vitamin A-related development in embryos.

PMWE effectively stimulates hair growth and is safer than minoxidil.

Thymus-derived Tregs, not peripherally-derived Tregs, primarily regulate type 1 diabetes in the NOD mouse model.

Rac1 is crucial for normal hair structure and pigmentation.

Overexpressing Dsg3 in mice skin causes excessive cell growth and abnormal skin development.

An artificial lipid barrier can restore hair growth in cases of SCD1 deficiency.

Removing PLCg1 from skin cells caused thicker oil glands and less hair in mice.