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The enzyme DHHC13 is essential for healthy hair and skin, and its deficiency leads to hair loss and skin problems.

A gene mutation in mice causes hair loss, weak bones, and protein buildup, showing how protein processing issues can lead to diseases.

ZDHHC13 is important for normal liver function and metabolism, affecting mitochondrial activity.

A mouse gene mutation increases the risk of skin cancer.

ZDHHC17 methylation may help treat or identify facial skin aging.

Mice with too much sPLA₂-IIA have hair loss and poor wound healing due to abnormal hair growth and stem cell depletion.

PP2Acα is essential for proper hair and skin development.

Genetic changes in mice help understand skin and hair disorders, aiding treatment development for acne and hair loss.

Targeting protein S-palmitoylation could lead to new skin disease treatments.

LRIG1 protein affects hair growth by regulating skin receptors, leading to hair loss when overexpressed.

New insights into cell communication in psoriasis suggest innovative drug treatments.

The environment around melanocyte stem cells is key for hair regeneration and color, with certain injuries affecting hair color and potential treatments for pigmentation disorders.

Melanoma's complexity requires personalized treatments due to key genetic mutations and tumor-initiating cells.

Goat genes show adaptation to environments and traits like body development, with differences among cashmere, feral, and milk-producing goats.

Camellia japonica extract may improve scalp health and promote hair growth.

AMPK activation may reduce melanoma risk in red-haired individuals.

DNA methylation changes in Tan sheep affect growth and fur traits.

A zinc-deficient diet stunted growth and affected organs in mice, with C57BL/6J mice showing more severe symptoms.

Hoxc13 gene is essential for hair, nail, and papilla development.

Mice with CBS deficiency are healthier on a low-methionine diet.

Knockout mice showed anemia and hair loss, suggesting other ways exist for iron absorption.

Iron deficiency causes hair loss by affecting hair differentiation and cycling.

A gene mutation in mice causes skin defects and early death.

Mice without the enzyme HSD17B3 still produce normal testosterone, suggesting they have different ways to make it compared to humans.

Mice without certain skin proteins had abnormal skin and hair development.

The SHJH hr mice with a mutated Hr gene show signs of faster skin aging due to poor antioxidative protection.

CD18-deficient mice developed psoriasis-like skin disease, useful for studying inflammatory skin disorders.

A gene deletion in mice causes weak protein, immune issues, hair loss, airway problems, and wasting disease.

Iron deficiency in mothers causes hair loss in their baby mice.