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Patients with the same genetic mutation for vitamin D-resistant rickets showed different symptoms but all improved with treatment except for hair loss.

Obese women with PCOS have lower vitamin D levels.

DDR-II hair follicle cells don't respond to 1, 25-dihydroxyvitamin D3, aiding diagnosis.

Tunisian children with hereditary vitamin D-resistant rickets showed improvement with calcium treatment, and new genetic mutations were identified.

The enzyme 25 Hydroxyvitamin D 1 α-Hydroxylase is essential for healthy skin and recovery after skin damage.

Vitamin D3 and its analogues affect skin cell growth and development depending on cell density, calcium levels, and serum presence.

Adding a specific gene to skin cells can help treat skin disorders like psoriasis.

Vitamin D supplements should be used cautiously in sarcoidosis patients, with regular monitoring to avoid high calcium levels.

Vitamin D3 can help improve hair growth by enhancing the function of specific skin cells and could be useful in hair regeneration treatments.

Vitamin D3 speeds up skin healing by helping skin stem cells grow and develop.

Vitamin D3 and its receptor help protect skin from UVB-related cancer.

Mice without the vitamin D receptor have bone issues and other health problems, suggesting vitamin D is important for preventing various diseases in humans.

Vitamin D receptor interacts with certain dietary components to help prevent diseases and regulate hair growth.

Vitamin D and its receptor are crucial for bone health and can be influenced by certain fats and curcumin.

VDDR I and II are genetic disorders affecting vitamin D use, causing rickets, with VDDR I treatable by vitamin D supplements and VDDR II needing high doses and calcium.

People with Alopecia Areata have lower vitamin D levels.

Vitamin D receptor helps control skin cell growth and hair development.

A girl had rickets due to a gene mutation affecting vitamin D response.

Vitamin D-dependent rickets Type II causes bone problems and hair loss, and doesn't improve with Vitamin D treatment.

Vitamin D receptor helps control hair growth genes in skin cells.

Vitamin D receptor can control the hairless gene linked to hair loss even without vitamin D.

Three siblings with a genetic form of rickets showed different symptoms of the disease.

Hair loss can be a key sign of a rare type of rickets when vitamin D treatment doesn't work and advanced tests aren't available.

A 2-year-old girl with a rare vitamin D disorder had rickets and hair loss, but treatment was ineffective due to poor compliance.

Vitamin D helps treat psoriasis by promoting skin cell differentiation and reducing growth.

Vitamin D may help protect against nonmelanoma skin cancer.

Higher androgen levels and site-specific AR expression cause sex-related skeletal differences, and certain steroids can boost AR expression and androgen effects in bone cells.

Lampreys have a functional vitamin D receptor that may help detoxify harmful substances.

Lack of vitamin D receptor increases skin tumor risk by boosting hedgehog signaling.