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A Chinese family had a child with a specific gene mutation causing vitamin D-resistant rickets, but the child improved with calcium and low-dose calcitriol.

Intravenous calcium therapy is a safe and effective treatment for a rare type of rickets.

Vitamin D3 and its analogues affect skin cell growth and development depending on cell density, calcium levels, and serum presence.

1 alpha,25-dihydroxy-vitamin D3 and calcipotriol speed up cell differentiation in hair follicles.

Active vitamin D3 might protect hair follicles from radiation damage.

Vitamin D3 and parathyroid hormone-related peptide may have important uses in skin treatments.

Vitamin D is made by the skin, helps control various body functions, and affects skin health and immunity.

Lithocholic acid helps hair growth and regeneration in alopecia by activating vitamin D receptors.

Some people have genetic mutations that make them resistant to vitamin D, leading to rickets even with enough vitamin D intake.

Vitamin D is important for skin cell growth and health, and its active form and receptor play key roles in skin and hair processes.

The vitamin D receptor helps regulate skin and hair health independently of its usual vitamin D ligand.

A specific gene mutation causes vitamin D-resistant rickets and hair loss.

Rubbing vitamin D3 on skin can help prevent hair loss from chemotherapy and slow breast tumor growth in mice.

A mutation in the vitamin D receptor causes severe resistance to vitamin D, affecting bone health but not hair growth.

The diphosphonate was the most effective at preventing calcification in rats.

A child with rickets and hair loss might have a rare type of rickets that doesn't improve much with usual vitamin D and calcium treatment.

Vitamin D compounds may help treat psoriasis by promoting skin cell differentiation.

A child with a rare type of rickets showed some improvement with high doses of vitamin D, but such conditions often respond poorly to treatment.

The R343H mutation in the VDR gene causes vitamin D-resistant rickets with alopecia by impairing specific gene activity.

New mutations in the VDR gene cause vitamin D-resistant rickets without hair loss.

A genetic mutation caused severe rickets and alopecia in an Indian patient, but high-dose calcium and phosphate treatment improved their condition.

The vitamin D receptor is crucial for bone health and affects various body systems, with mutations potentially leading to disease.

Vitamin D is important for regulating calcium, phosphate, and hair health.

Patients with the same genetic mutation for vitamin D-resistant rickets showed different symptoms but all improved with treatment except for hair loss.

Two sisters with rickets and hair loss had a genetic issue with vitamin D processing, and only improved when given phosphorus supplements.

Vitamin D receptor and mediator 1 are crucial for healthy skin and hair growth.

A new gene mutation causes vitamin D resistance and hair loss in two unrelated girls.

A unique gene mutation causes vitamin D-resistant rickets without causing hair loss.