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Melanocyte pathology requires keratinocyte hyperplasia and regulation dysfunction.

Retinoic acid affects male and female muscle energy use and function differently.

Curcuma aeruginosa Roxb. may help treat hair loss by affecting specific biological pathways.

TTNPB helps turn stem cells into neural stem cells, improving depression-like behaviors in rats.

MCP@G improves diabetic wound healing by reducing stress and promoting tissue repair.

Annurca apple extract promotes hair growth by changing hair follicle metabolism to boost keratin production.

MicroRNA-302 helps improve the conversion of body cells into stem cells by blocking NR2F2.

CD34⁺ cells help heal damaged limbs by promoting blood vessel growth.

New treatments may restore cancer-blocking proteins, slow prostate cancer, identify drug targets, and potentially regrow hair.

New drugs and therapies targeting specific pathways show promise in treating advanced prostate cancer.

The scalp fat tissue of men with hair loss shows changes in gene activity that may contribute to their condition.

Applying certain inhibitors to the skin can promote hair growth without harming cells.

EGM2 and SOX2 help form beige adipocytes by maintaining ASC immaturity and activating brown adipocytes.

AcSDKP may help prevent skin and hair aging and promote their growth.

Increasing SSAT makes skin more prone to cancer.

Akkermansia muciniphila may help prevent testosterone-related hair loss.

Phospholipids help plant proteins move by regulating receptor interactions.

A new therapy sped up wound healing and reduced scarring in diabetic rats.

Using drugs to activate the Wnt/β-catenin pathway has potential for treating diseases but also presents challenges.

Targeting the p300/AR axis may help treat polycystic ovary syndrome.

Removing Gsdma1/2/3 genes reduces skin cell overgrowth by blocking a specific cell pathway.

Peptide nanoparticles can effectively deliver CRISPR-Cas9 to target KRAS mutations in cancer.

Certain electric currents can promote hair growth by activating specific cell pathways.

Tideglusib helps heal wounds in older skin.

Targeting amphiregulin may improve treatment for fibrosis and cancer.

PROTAC technology shows promise for cancer treatment but needs more effective E3 ligase recruiters.

Poncirin is a promising inhibitor of Janus Kinase 3, potentially better than tofacitinib.

Stem cells control their future role by changing ERK signal timing, affecting tissue regeneration and cancer.

The L457(3.43)R mutation in the human lutropin receptor causes increased activity and hormone insensitivity, leading to precocious puberty.

Activin helps skin growth and healing mainly through stromal cells and affects keratinocytes based on its amount.