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The document concludes that a systematic approach is crucial to identify causes of androgen excess in women beyond the most common cause, Polycystic ovary syndrome (PCOS).

Seasonal changes affect gene activity linked to hair growth in Angora goats, influencing mohair quality.

Flutamide caused damage to male rat reproductive organs and may affect fertility.

Early investigation and a team approach are crucial for managing primary amenorrhea effectively.

The new rodent model successfully mimics non-lean human PCOS symptoms.

Pregnancy increases certain GABA_A receptors in rat brains, influenced by steroid levels.

Melatonin helped some Pomeranian dogs regrow hair, but it wasn't linked to estrogen receptors.

A new compound was made that might help treat diseases related to male hormones.

Certain adrenal hormones can strongly stimulate oil gland growth in hamster skin, similar to male hormones.

Cyproterone acetate is a safe treatment that causes mild feminizing effects and is more effective with added estrogens.

Prostate-specific antigen may be a new marker for excess male hormones in women with polycystic ovary syndrome.

Estradiol stops hair growth in mice, but an antagonist can reverse this effect.

Affected males are born with ambiguous genitalia, raised as females, but develop male traits at puberty due to enzyme deficiency.

Early hormones shape sex-specific differences in rat glands.

A 31-year-old woman was diagnosed with Complete Androgen Insensitivity Syndrome much later than usual, leading to a call for earlier detection and treatment guidelines.

A rare ovarian tumor caused high testosterone and excess hair in a woman, which was resolved after tumor removal.

Estrogen receptors affect hair growth, with ER beta slowing down the hair cycle changes caused by ER alpha.

The document suggests "anisotrichosis" as a new term for hair thickness variation in common baldness.

A subtle androgen receptor abnormality can allow normal male development and sometimes fertility despite partial androgen resistance.

Testosterone increases certain proteins in the uterus, reducing fluid volume.

Rodent models of PCOS show some hormone changes similar to humans, but also have key differences.

The steroids allopregnanolone and allotetrahydrodeoxycorticosteron worsened absence seizures in rats.